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Tappei Takada

Showing results (141-150 of 153) with videos related to

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Scientific Reports|January 21, 2014
ABCG2 dysfunction causes hyperuricemia due to both renal urate underexcretion and renal urate overloadHirotaka Matsuo, Akiyoshi Nakayama, Masayuki Sakiyama, et al.
Scientific Reports|June 19, 2013
Common dysfunctional variants in ABCG2 are a major cause of early-onset goutHirotaka Matsuo, Kimiyoshi Ichida, Tappei Takada, et al.
Annals of the Rheumatic Diseases|November 30, 2019
Dysfunctional missense variant of <i>OAT10/SLC22A13</i> decreases gout risk and serum uric acid levelsToshihide Higashino, Keito Morimoto, Hirofumi Nakaoka, et al.
RMD Open|December 12, 2017
Multiple common and rare variants of <i>ABCG2</i> cause goutToshihide Higashino, Tappei Takada, Hirofumi Nakaoka, et al.
Science Translational Medicine|April 7, 2010
Common defects of ABCG2, a high-capacity urate exporter, cause gout: a function-based genetic analysis in a Japanese populationHirotaka Matsuo, Tappei Takada, Kimiyoshi Ichida, et al.
Japanese Journal of Clinical Oncology|July 9, 2026
Triplet therapy with darolutamide and docetaxel for metastatic castration-sensitive prostate cancer: a multicenter real-world studyIbuki Tsuru, Satoru Taguchi, Ryo Tanaka, et al.
International Journal of Urology : Official Journal of the Japanese Urological Association|November 22, 2024
Physical, but not laboratory, treatment-related adverse events are associated with favorable outcomes of enfortumab vedotin for advanced urothelial carcinoma: A landmark analysisSatoru Taguchi, Taketo Kawai, Yoshiki Ambe, et al.
Annals of the Rheumatic Diseases|February 4, 2015
Genome-wide association study of clinically defined gout identifies multiple risk loci and its association with clinical subtypesHirotaka Matsuo, Ken Yamamoto, Hirofumi Nakaoka, et al.
Annals of the Rheumatic Diseases|December 1, 2016
GWAS of clinically defined gout and subtypes identifies multiple susceptibility loci that include urate transporter genesAkiyoshi Nakayama, Hirofumi Nakaoka, Ken Yamamoto, et al.
Annals of the Rheumatic Diseases|July 11, 2019
Genome-wide association study revealed novel loci which aggravate asymptomatic hyperuricaemia into goutYusuke Kawamura, Hirofumi Nakaoka, Akiyoshi Nakayama, et al.
Pageof 16

Showing results (141-150 of 153) with videos related to

Sort By:
Pageof 16
Scientific Reports|January 21, 2014
ABCG2 dysfunction causes hyperuricemia due to both renal urate underexcretion and renal urate overloadHirotaka Matsuo, Akiyoshi Nakayama, Masayuki Sakiyama, et al.
Scientific Reports|June 19, 2013
Common dysfunctional variants in ABCG2 are a major cause of early-onset goutHirotaka Matsuo, Kimiyoshi Ichida, Tappei Takada, et al.
Annals of the Rheumatic Diseases|November 30, 2019
Dysfunctional missense variant of <i>OAT10/SLC22A13</i> decreases gout risk and serum uric acid levelsToshihide Higashino, Keito Morimoto, Hirofumi Nakaoka, et al.
RMD Open|December 12, 2017
Multiple common and rare variants of <i>ABCG2</i> cause goutToshihide Higashino, Tappei Takada, Hirofumi Nakaoka, et al.
Science Translational Medicine|April 7, 2010
Common defects of ABCG2, a high-capacity urate exporter, cause gout: a function-based genetic analysis in a Japanese populationHirotaka Matsuo, Tappei Takada, Kimiyoshi Ichida, et al.
Japanese Journal of Clinical Oncology|July 9, 2026
Triplet therapy with darolutamide and docetaxel for metastatic castration-sensitive prostate cancer: a multicenter real-world studyIbuki Tsuru, Satoru Taguchi, Ryo Tanaka, et al.
International Journal of Urology : Official Journal of the Japanese Urological Association|November 22, 2024
Physical, but not laboratory, treatment-related adverse events are associated with favorable outcomes of enfortumab vedotin for advanced urothelial carcinoma: A landmark analysisSatoru Taguchi, Taketo Kawai, Yoshiki Ambe, et al.
Annals of the Rheumatic Diseases|February 4, 2015
Genome-wide association study of clinically defined gout identifies multiple risk loci and its association with clinical subtypesHirotaka Matsuo, Ken Yamamoto, Hirofumi Nakaoka, et al.
Annals of the Rheumatic Diseases|December 1, 2016
GWAS of clinically defined gout and subtypes identifies multiple susceptibility loci that include urate transporter genesAkiyoshi Nakayama, Hirofumi Nakaoka, Ken Yamamoto, et al.
Annals of the Rheumatic Diseases|July 11, 2019
Genome-wide association study revealed novel loci which aggravate asymptomatic hyperuricaemia into goutYusuke Kawamura, Hirofumi Nakaoka, Akiyoshi Nakayama, et al.
Pageof 16