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Endocrinology|December 19, 2002
p53 Is required for 1,25-dihydroxyvitamin D3-induced G0 arrest but is not required for G1 accumulation or apoptosis of LNCaP prostate cancer cellsTara C Polek, LaMonica V Stewart, Elizabeth J Ryu, et al.Biochemical and Biophysical Research Communications|March 15, 2006
The TNF receptor, RELT, binds SPAK and uses it to mediate p38 and JNK activationTara C Polek, Moshe Talpaz, Taly Spivak-KroizmanBiochemical and Biophysical Research Communications|September 5, 2006
TRAIL-induced cleavage and inactivation of SPAK sensitizes cells to apoptosisTara C Polek, Moshe Talpaz, Taly R Spivak-KroizmanThe Journal of Biological Chemistry|June 10, 2003
TWEAK mediates signal transduction and differentiation of RAW264.7 cells in the absence of Fn14/TweakR. Evidence for a second TWEAK receptorTara C Polek, Moshe Talpaz, Bryant G Darnay, et al.The Prostate|March 26, 2004
1alpha,25-dihydroxyvitamin D3 induced growth inhibition of PC-3 prostate cancer cells requires an active transforming growth factor beta signaling pathwayShalini Murthy, Nancy L WeigelThe International Journal of Biochemistry & Cell Biology|May 10, 2011
Regulation of progesterone receptor activity by cyclin dependent kinases 1 and 2 occurs in part by phosphorylation of the SRC-1 carboxyl-terminusNicole L Moore, Nancy L WeigelExperimental Biology and Medicine (Maywood, N.J.)|March 27, 2004
Vitamin D and prostate cancerLaMonica V Stewart, Nancy L WeigelThe Prostate|January 15, 2005
Role of insulin-like growth factor binding proteins in 1alpha,25-dihydroxyvitamin D(3)-induced growth inhibition of human prostate cancer cellsLamonica V Stewart, Nancy L WeigelEndocrinology|February 12, 2010
1{alpha},25-Dihydroxyvitamin D3 inhibits growth of VCaP prostate cancer cells despite inducing the growth-promoting TMPRSS2:ERG gene fusionMichele N Washington, Nancy L WeigelPageof 8