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Diabetes|March 16, 2007
Downregulation of GLP-1 and GIP receptor expression by hyperglycemia: possible contribution to impaired incretin effects in diabetesGang Xu, Hideaki Kaneto, D Ross Laybutt, et al.Diabetes|June 19, 2013
PDX1 in ducts is not required for postnatal formation of β-cells but is necessary for their subsequent maturationLili Guo, Akari Inada, Cristina Aguayo-Mazzucato, et al.Endocrinology|April 4, 2015
Reprogramming Mouse Cells With a Pancreatic Duct Phenotype to Insulin-Producing β-Like CellsTakatsugu Yamada, Claudia Cavelti-Weder, Francisco Caballero, et al.The Journal of Clinical Endocrinology and Metabolism|December 13, 2007
Gene expression of purified beta-cell tissue obtained from human pancreas with laser capture microdissectionLorella Marselli, Jeffrey Thorne, Yu-Bae Ahn, et al.Diabetes|November 14, 2013
Occurrence of spontaneous pancreatic lesions in normal and diabetic rats: a potential confounding factor in the nonclinical assessment of GLP-1-based therapiesKristina D Chadwick, Anthony M Fletcher, M Cecilia Parrula, et al.Cellular Reprogramming|December 2, 2014
β-Cell differentiation of human pancreatic duct-derived cells after in vitro expansionElisa Corritore, Erica Dugnani, Valentina Pasquale, et al.Cell Metabolism|June 4, 2019
Acceleration of β Cell Aging Determines Diabetes and Senolysis Improves Disease OutcomesCristina Aguayo-Mazzucato, Joshua Andle, Terrence B Lee, et al.Aging|July 9, 2026
TM4SF1 is a surface marker of senescent pancreatic β-cellsAna Beathriz Leite Lorente, Sergio Vazquez, Kanako Iwasaki, et al.The American Journal of Pathology|July 29, 2005
Establishment of a diabetic mouse model with progressive diabetic nephropathyAkari Inada, Kojiro Nagai, Hidenori Arai, et al.Plos One|November 9, 2013
Sustained NF-κB activation and inhibition in β-cells have minimal effects on function and islet transplant outcomesAileen J F King, Yongjing Guo, Dongsheng Cai, et al.Pageof 15