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Nature Communications|January 10, 2019
ATM orchestrates the DNA-damage response to counter toxic non-homologous end-joining at broken replication forksGabriel Balmus, Domenic Pilger, Julia Coates, et al.Haematologica|April 5, 2015
Targeting the Ataxia Telangiectasia Mutated-null phenotype in chronic lymphocytic leukemia with pro-oxidantsAngelo Agathanggelou, Victoria J Weston, Tracey Perry, et al.Blood|February 4, 2016
Clinical impact of clonal and subclonal TP53, SF3B1, BIRC3, NOTCH1, and ATM mutations in chronic lymphocytic leukemiaFerran Nadeu, Julio Delgado, Cristina Royo, et al.Medchemcomm|September 21, 2020
Derivatisation of parthenolide to address chemoresistant chronic lymphocytic leukaemiaXingjian Li, Daniel T Payne, Badarinath Ampolu, et al.Haematologica|June 6, 2024
Genome-scale clustered regularly interspaced short palindromic repeats screen identifies nucleotide metabolism as an actionable therapeutic vulnerability in diffuse large B-cell lymphomaNicholas Davies, Tegan Francis, Ceri Oldreive, et al.Blood|December 4, 2019
Integrative analysis of spontaneous CLL regression highlights genetic and microenvironmental interdependency in CLLMarwan Kwok, Ceri Oldreive, Andy C Rawstron, et al.Nature Genetics|July 31, 2023
SF3B1 hotspot mutations confer sensitivity to PARP inhibition by eliciting a defective replication stress responsePhilip Bland, Harry Saville, Patty T Wai, et al.Nature Communications|May 14, 2025
Inherited deficiency of DIAPH1 identifies a DNA double strand break repair pathway regulated by γ-actinBeth L Woodward, Sudipta Lahiri, Anoop S Chauhan, et al.Pageof 6