Showing results (1-10 of 85) with videos related to
Sort By:
Pageof 9
Biological & Pharmaceutical Bulletin|August 4, 2015
Cell Death in the Cardiac MyocyteTetsuro Marunouchi, Kouichi TanonakaBiomedicine & Pharmacotherapy = Biomedecine & Pharmacotherapie|October 31, 2018
Effects of cardiosphere-derived cell transplantation on cardiac mitochondrial oxygen consumption after myocardial infarction in ratsTetsuro Marunouchi, Emi Yano, Kouichi TanonakaBiological & Pharmaceutical Bulletin|January 11, 2019
Histone Deacetylase Inhibitor SAHA Treatment Prevents the Development of Heart Failure after Myocardial Infarction via an Induction of Heat-Shock Proteins in RatsShiho Nagata, Tetsuro Marunouchi, Kouichi TanonakaBiological & Pharmaceutical Bulletin|January 4, 2026
Tanshinone VI Attenuates the Development of Heart Failure with Preserved Ejection Fraction in MiceTetsuro Marunouchi, Yuji Kawahara, Kouichi TanonakaJournal of Molecular and Cellular Cardiology|December 25, 2018
Heat-shock protein 90 modulates cardiac ventricular hypertrophy via activation of MAPK pathwayShoko Tamura, Tetsuro Marunouchi, Kouichi TanonakaBiological & Pharmaceutical Bulletin|August 27, 2025
Tanshinone VI Induces Cardiac Differentiation of Cardiosphere-Derived Cells Prepared from Rat HeartsYuji Kawahara, Tetsuro Marunouchi, Kouichi TanonakaBiological & Pharmaceutical Bulletin|August 2, 2013
Possible involvement of phosphorylated heat-shock factor-1 in changes in heat shock protein 72 induction in the failing rat heart following myocardial infarctionTetsuro Marunouchi, Mao Murata, Norio Takagi, et al.Biological & Pharmaceutical Bulletin|April 7, 2024
Angiotensin II Is Involved in MLKL Activation During the Development of Heart Failure Following Myocardial Infarction in RatsTetsuro Marunouchi, Sumika Onda, Minami Kurasawa, et al.Journal of Molecular and Cellular Cardiology Plus|March 26, 2025
Gemfibrozil mitigates caspase-11-driven myocardial pyroptosis in ischemia/reperfusion injury in miceTetsuro Marunouchi, Mayu Kyono, Naoko Kikuchi, et al.Journal of Pharmacological Sciences|August 4, 2019
Transplantation of cardiac Sca-1-positive cells rather than c-Kit-positive cells preserves mitochondrial oxygen consumption of the viable myocardium following myocardial infarction in ratsTetsuro Marunouchi, Kyohei Sasaki, Emi Yano, et al.Pageof 9