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Development (Cambridge, England)|April 13, 2016
Thyroid follicle development requires Smad1/5- and endothelial cell-dependent basement membrane assemblyMylah Villacorte, Anne-Sophie Delmarcelle, Manon Lernoux, et al.
The Journal of Clinical Endocrinology and Metabolism|October 18, 2007
Pendred syndrome in two Galician families: insights into clinical phenotypes through cellular, genetic, and molecular studiesFernando Palos, María E R García-Rendueles, David Araujo-Vilar, et al.
Nature Communications|November 17, 2022
Transplantable human thyroid organoids generated from embryonic stem cells to rescue hypothyroidismMírian Romitti, Adrien Tourneur, Barbara de Faria da Fonseca, et al.
Cancer Cell|January 16, 2026
A context-augmented large language model for accurate precision oncology medicine recommendationsHyeji Jun, Yutaro Tanaka, Shreya Johri, et al.
Thyroid : Official Journal of the American Thyroid Association|March 7, 2023
Iodotyrosines Are Biomarkers for Preclinical Stages of Iodine-Deficient Hypothyroidism in Dehal1-Knockout MiceCristian González-Guerrero, Marco Borsò, Pouya Alikhani, et al.
JAMA Oncology|October 24, 2024
Dual Immune Checkpoint Inhibition in Patients With Aggressive Thyroid Carcinoma: A Phase 2 Nonrandomized Clinical TrialKartik Sehgal, Theodora Pappa, Kee-Young Shin, et al.
Cancer Discovery|December 15, 2020
SWI/SNF Complex Mutations Promote Thyroid Tumor Progression and Insensitivity to Redifferentiation TherapiesMahesh Saqcena, Luis Javier Leandro-Garcia, Jesper L V Maag, et al.
Cell Stem Cell|May 21, 2017
Modeling Psychomotor Retardation using iPSCs from MCT8-Deficient Patients Indicates a Prominent Role for the Blood-Brain BarrierGad D Vatine, Abraham Al-Ahmad, Bianca K Barriga, et al.
Thyroid : Official Journal of the American Thyroid Association|May 11, 2018
NFE2-Related Transcription Factor 2 Coordinates Antioxidant Defense with Thyroglobulin Production and Iodination in the Thyroid GlandPanos G Ziros, Ioannis G Habeos, Dionysios V Chartoumpekis, et al.
JCI Insight|October 19, 2018
Homozygous loss-of-function mutations in SLC26A7 cause goitrous congenital hypothyroidismHakan Cangul, Xiao-Hui Liao, Erik Schoenmakers, et al.
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