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Theodore R Cummins

Showing results (91-100 of 109) with videos related to

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Molecular Pain|September 23, 2008
Paroxysmal extreme pain disorder M1627K mutation in human Nav1.7 renders DRG neurons hyperexcitableSulayman D Dib-Hajj, Mark Estacion, Brian W Jarecki, et al.
The Journal of Biological Chemistry|April 20, 2012
Ca2+/calmodulin-dependent protein kinase II (CaMKII) regulates cardiac sodium channel NaV1.5 gating by multiple phosphorylation sitesNicole M Ashpole, Anthony W Herren, Kenneth S Ginsburg, et al.
Frontiers in Cellular Neuroscience|June 29, 2019
Hedgehog Pathway Activation Alters Ciliary Signaling in Primary Hypothalamic CulturesRuchi Bansal, Staci E Engle, Patrick J Antonellis, et al.
The Journal of Pain|April 25, 2006
The role of sodium channels in chronic inflammatory and neuropathic painRon Amir, Charles E Argoff, Gary J Bennett, et al.
Heart Rhythm|February 8, 2018
Calcium/calmodulin-dependent protein kinase II regulation of I<sub>Ks</sub> during sustained β-adrenergic receptor stimulationTyler Shugg, Derrick E Johnson, Minghai Shao, et al.
Molecular Pain|September 27, 2015
Navβ4 regulates fast resurgent sodium currents and excitability in sensory neuronsCindy Barbosa, Zhi-Yong Tan, Ruizhong Wang, et al.
Biopolymers|June 9, 2016
t-boc synthesis of huwentoxin-i through native chemical ligation incorporating a trifluoromethanesulfonic acid cleavage strategyParashar Thapa, Chino C Cabalteja, Edwin E Philips, et al.
Glia|January 8, 2026
Müller Glial Kir4.1 Channel Dysfunction in APOE4-KI Model of Alzheimer's DiseaseSurabhi D Abhyankar, Yucheng Xiao, Neha Mahajan, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|May 23, 2014
Tetrodotoxin-resistant sodium channels in sensory neurons generate slow resurgent currents that are enhanced by inflammatory mediatorsZhi-Yong Tan, Andrew D Piekarz, Birgit T Priest, et al.
The Journal of Biological Chemistry|June 12, 2010
In silico docking and electrophysiological characterization of lacosamide binding sites on collapsin response mediator protein-2 identifies a pocket important in modulating sodium channel slow inactivationYuying Wang, Joel M Brittain, Brian W Jarecki, et al.
Pageof 11

Showing results (91-100 of 109) with videos related to

Sort By:
Pageof 11
Molecular Pain|September 23, 2008
Paroxysmal extreme pain disorder M1627K mutation in human Nav1.7 renders DRG neurons hyperexcitableSulayman D Dib-Hajj, Mark Estacion, Brian W Jarecki, et al.
The Journal of Biological Chemistry|April 20, 2012
Ca2+/calmodulin-dependent protein kinase II (CaMKII) regulates cardiac sodium channel NaV1.5 gating by multiple phosphorylation sitesNicole M Ashpole, Anthony W Herren, Kenneth S Ginsburg, et al.
Frontiers in Cellular Neuroscience|June 29, 2019
Hedgehog Pathway Activation Alters Ciliary Signaling in Primary Hypothalamic CulturesRuchi Bansal, Staci E Engle, Patrick J Antonellis, et al.
The Journal of Pain|April 25, 2006
The role of sodium channels in chronic inflammatory and neuropathic painRon Amir, Charles E Argoff, Gary J Bennett, et al.
Heart Rhythm|February 8, 2018
Calcium/calmodulin-dependent protein kinase II regulation of I<sub>Ks</sub> during sustained β-adrenergic receptor stimulationTyler Shugg, Derrick E Johnson, Minghai Shao, et al.
Molecular Pain|September 27, 2015
Navβ4 regulates fast resurgent sodium currents and excitability in sensory neuronsCindy Barbosa, Zhi-Yong Tan, Ruizhong Wang, et al.
Biopolymers|June 9, 2016
t-boc synthesis of huwentoxin-i through native chemical ligation incorporating a trifluoromethanesulfonic acid cleavage strategyParashar Thapa, Chino C Cabalteja, Edwin E Philips, et al.
Glia|January 8, 2026
Müller Glial Kir4.1 Channel Dysfunction in APOE4-KI Model of Alzheimer's DiseaseSurabhi D Abhyankar, Yucheng Xiao, Neha Mahajan, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|May 23, 2014
Tetrodotoxin-resistant sodium channels in sensory neurons generate slow resurgent currents that are enhanced by inflammatory mediatorsZhi-Yong Tan, Andrew D Piekarz, Birgit T Priest, et al.
The Journal of Biological Chemistry|June 12, 2010
In silico docking and electrophysiological characterization of lacosamide binding sites on collapsin response mediator protein-2 identifies a pocket important in modulating sodium channel slow inactivationYuying Wang, Joel M Brittain, Brian W Jarecki, et al.
Pageof 11