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Theodore R Cummins

Showing results (31-40 of 109) with videos related to

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Brain : a Journal of Neurology|June 9, 2016
Aberrant epilepsy-associated mutant Nav1.6 sodium channel activity can be targeted with cannabidiolReesha R Patel, Cindy Barbosa, Tatiana Brustovetsky, et al.
Investigative Ophthalmology & Visual Science|May 3, 2019
The Diurnal Rhythm of Insulin Receptor Substrate-1 (IRS-1) and Kir4.1 in Diabetes: Implications for a Clock Gene Bmal1Qianyi Luo, Yucheng Xiao, Alpha Alex, et al.
The Journal of Clinical Investigation|December 30, 2009
Human voltage-gated sodium channel mutations that cause inherited neuronal and muscle channelopathies increase resurgent sodium currentsBrian W Jarecki, Andrew D Piekarz, James O Jackson, et al.
The Journal of Physiology|December 1, 2010
Nav1.7 mutations associated with paroxysmal extreme pain disorder, but not erythromelalgia, enhance Navbeta4 peptide-mediated resurgent sodium currentsJonathan W Theile, Brian W Jarecki, Andrew D Piekarz, et al.
Molecular Pharmacology|September 22, 2010
The tarantula toxins ProTx-II and huwentoxin-IV differentially interact with human Nav1.7 voltage sensors to inhibit channel activation and inactivationYucheng Xiao, Kenneth Blumenthal, James O Jackson, et al.
The Journal of Physiology|July 5, 2008
Paroxysmal extreme pain disorder mutations within the D3/S4-S5 linker of Nav1.7 cause moderate destabilization of fast inactivationBrian W Jarecki, Patrick L Sheets, James O Jackson, et al.
Novartis Foundation Symposium|January 5, 2002
Diverse functions and dynamic expression of neuronal sodium channelsStephen G Waxman, Theodore R Cummins, Joel A Black, et al.
Trends in Neurosciences|October 24, 2007
From genes to pain: Na v 1.7 and human pain disordersSulayman D Dib-Hajj, Theodore R Cummins, Joel A Black, et al.
Scientific Reports|August 15, 2013
Structural basis for the modulation of the neuronal voltage-gated sodium channel NaV1.6 by calmodulinVishnu Priyanka Reddy Chichili, Yucheng Xiao, J Seetharaman, et al.
Biomedicines|September 27, 2025
Voltage-Gated Sodium Channel Dysfunction in Epilepsy: Zebrafish Models for TherapeuticsAngela Gyamfi, Priyadharshini Manikandan, William A Cisneros, et al.
Pageof 11

Showing results (31-40 of 109) with videos related to

Sort By:
Pageof 11
Brain : a Journal of Neurology|June 9, 2016
Aberrant epilepsy-associated mutant Nav1.6 sodium channel activity can be targeted with cannabidiolReesha R Patel, Cindy Barbosa, Tatiana Brustovetsky, et al.
Investigative Ophthalmology & Visual Science|May 3, 2019
The Diurnal Rhythm of Insulin Receptor Substrate-1 (IRS-1) and Kir4.1 in Diabetes: Implications for a Clock Gene Bmal1Qianyi Luo, Yucheng Xiao, Alpha Alex, et al.
The Journal of Clinical Investigation|December 30, 2009
Human voltage-gated sodium channel mutations that cause inherited neuronal and muscle channelopathies increase resurgent sodium currentsBrian W Jarecki, Andrew D Piekarz, James O Jackson, et al.
The Journal of Physiology|December 1, 2010
Nav1.7 mutations associated with paroxysmal extreme pain disorder, but not erythromelalgia, enhance Navbeta4 peptide-mediated resurgent sodium currentsJonathan W Theile, Brian W Jarecki, Andrew D Piekarz, et al.
Molecular Pharmacology|September 22, 2010
The tarantula toxins ProTx-II and huwentoxin-IV differentially interact with human Nav1.7 voltage sensors to inhibit channel activation and inactivationYucheng Xiao, Kenneth Blumenthal, James O Jackson, et al.
The Journal of Physiology|July 5, 2008
Paroxysmal extreme pain disorder mutations within the D3/S4-S5 linker of Nav1.7 cause moderate destabilization of fast inactivationBrian W Jarecki, Patrick L Sheets, James O Jackson, et al.
Novartis Foundation Symposium|January 5, 2002
Diverse functions and dynamic expression of neuronal sodium channelsStephen G Waxman, Theodore R Cummins, Joel A Black, et al.
Trends in Neurosciences|October 24, 2007
From genes to pain: Na v 1.7 and human pain disordersSulayman D Dib-Hajj, Theodore R Cummins, Joel A Black, et al.
Scientific Reports|August 15, 2013
Structural basis for the modulation of the neuronal voltage-gated sodium channel NaV1.6 by calmodulinVishnu Priyanka Reddy Chichili, Yucheng Xiao, J Seetharaman, et al.
Biomedicines|September 27, 2025
Voltage-Gated Sodium Channel Dysfunction in Epilepsy: Zebrafish Models for TherapeuticsAngela Gyamfi, Priyadharshini Manikandan, William A Cisneros, et al.
Pageof 11