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Thomas A Cooper

Showing results (51-60 of 106) with videos related to

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Molecular Cell|April 5, 2002
Dynamic antagonism between ETR-3 and PTB regulates cell type-specific alternative splicingNicolas Charlet-B, Penny Logan, Gopal Singh, et al.
Human Molecular Genetics|May 16, 2020
Increased nuclear but not cytoplasmic activities of CELF1 protein leads to muscle wastingDiana C Cox, Xiangnan Guan, Zheng Xia, et al.
Nucleic Acids Research|February 20, 2004
ETR-3 and CELF4 protein domains required for RNA binding and splicing activity in vivoGopal Singh, Nicolas Charlet-B, Jin Han, et al.
Molecular Cell|October 16, 2007
Increased steady-state levels of CUGBP1 in myotonic dystrophy 1 are due to PKC-mediated hyperphosphorylationN Muge Kuyumcu-Martinez, Guey-Shin Wang, Thomas A Cooper
Nucleic Acids Research|November 27, 2010
Identification of MBNL1 and MBNL3 domains required for splicing activation and repressionIoannis Grammatikakis, Young-Hwa Goo, Gloria V Echeverria, et al.
Human Molecular Genetics|August 10, 2024
Rescue of Scn5a mis-splicing does not improve the structural and functional heart defects of a DM1 heart mouse modelLarissa Nitschke, Rong-Chi Hu, Andrew N Miller, et al.
The Journal of Biological Chemistry|February 6, 2004
CELF6, a member of the CELF family of RNA-binding proteins, regulates muscle-specific splicing enhancer-dependent alternative splicingAndrea N Ladd, Nicole H Nguyen, Kavin Malhotra, et al.
Human Molecular Genetics|April 22, 2005
Transgenic mice expressing CUG-BP1 reproduce splicing mis-regulation observed in myotonic dystrophyThai H Ho, Donnie Bundman, Dawna L Armstrong, et al.
ACS Chemical Biology|August 31, 2017
BNA<sup>NC</sup> Gapmers Revert Splicing and Reduce RNA Foci with Low Toxicity in Myotonic Dystrophy CellsKassie S Manning, Ashish N Rao, Miguel Castro, et al.
Cell Reports|November 17, 2016
Alternative Splicing of Four Trafficking Genes Regulates Myofiber Structure and Skeletal Muscle PhysiologyJimena Giudice, James A Loehr, George G Rodney, et al.
Pageof 11

Showing results (51-60 of 106) with videos related to

Sort By:
Pageof 11
Molecular Cell|April 5, 2002
Dynamic antagonism between ETR-3 and PTB regulates cell type-specific alternative splicingNicolas Charlet-B, Penny Logan, Gopal Singh, et al.
Human Molecular Genetics|May 16, 2020
Increased nuclear but not cytoplasmic activities of CELF1 protein leads to muscle wastingDiana C Cox, Xiangnan Guan, Zheng Xia, et al.
Nucleic Acids Research|February 20, 2004
ETR-3 and CELF4 protein domains required for RNA binding and splicing activity in vivoGopal Singh, Nicolas Charlet-B, Jin Han, et al.
Molecular Cell|October 16, 2007
Increased steady-state levels of CUGBP1 in myotonic dystrophy 1 are due to PKC-mediated hyperphosphorylationN Muge Kuyumcu-Martinez, Guey-Shin Wang, Thomas A Cooper
Nucleic Acids Research|November 27, 2010
Identification of MBNL1 and MBNL3 domains required for splicing activation and repressionIoannis Grammatikakis, Young-Hwa Goo, Gloria V Echeverria, et al.
Human Molecular Genetics|August 10, 2024
Rescue of Scn5a mis-splicing does not improve the structural and functional heart defects of a DM1 heart mouse modelLarissa Nitschke, Rong-Chi Hu, Andrew N Miller, et al.
The Journal of Biological Chemistry|February 6, 2004
CELF6, a member of the CELF family of RNA-binding proteins, regulates muscle-specific splicing enhancer-dependent alternative splicingAndrea N Ladd, Nicole H Nguyen, Kavin Malhotra, et al.
Human Molecular Genetics|April 22, 2005
Transgenic mice expressing CUG-BP1 reproduce splicing mis-regulation observed in myotonic dystrophyThai H Ho, Donnie Bundman, Dawna L Armstrong, et al.
ACS Chemical Biology|August 31, 2017
BNA<sup>NC</sup> Gapmers Revert Splicing and Reduce RNA Foci with Low Toxicity in Myotonic Dystrophy CellsKassie S Manning, Ashish N Rao, Miguel Castro, et al.
Cell Reports|November 17, 2016
Alternative Splicing of Four Trafficking Genes Regulates Myofiber Structure and Skeletal Muscle PhysiologyJimena Giudice, James A Loehr, George G Rodney, et al.
Pageof 11