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Thomas A Cooper

Showing results (71-80 of 106) with videos related to

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The Journal of Clinical Investigation|September 8, 2007
Elevation of RNA-binding protein CUGBP1 is an early event in an inducible heart-specific mouse model of myotonic dystrophyGuey-Shin Wang, Debra L Kearney, Mariella De Biasi, et al.
Human Molecular Genetics|January 7, 2010
Heart-specific overexpression of CUGBP1 reproduces functional and molecular abnormalities of myotonic dystrophy type 1Misha Koshelev, Satyam Sarma, Roger E Price, et al.
Iscience|October 30, 2024
Alternatively spliced MAP4 isoforms have key roles in maintaining microtubule organization and skeletal muscle functionLathan Lucas, Larissa Nitschke, Brandon Nguyen, et al.
The International Journal of Biochemistry & Cell Biology|October 15, 2018
Modulation of alternative splicing of trafficking genes by genome editing reveals functional consequences in muscle biologyR Eric Blue, Amrita Koushik, Nichlas M Engels, et al.
Cell Reports|January 14, 2014
The Mef2 transcription network is disrupted in myotonic dystrophy heart tissue, dramatically altering miRNA and mRNA expressionAuinash Kalsotra, Ravi K Singh, Priyatansh Gurha, et al.
Proceedings of the National Academy of Sciences of the United States of America|February 15, 2008
Expanded CTG repeats within the DMPK 3' UTR causes severe skeletal muscle wasting in an inducible mouse model for myotonic dystrophyJames P Orengo, Pierre Chambon, Daniel Metzger, et al.
Human Molecular Genetics|August 30, 2007
Expression, localization and tau exon 10 splicing activity of the brain RNA-binding protein TNRC4J Paul Chapple, Karen Anthony, Teresa Rodriguez Martin, et al.
Elife|August 23, 2017
Extensive alternative splicing transitions during postnatal skeletal muscle development are required for calcium handling functionsAmy E Brinegar, Zheng Xia, James Anthony Loehr, et al.
Molecular and Cellular Biology|October 17, 2024
Small Molecule Screening Identifies HSP90 as a Modifier of RNA Foci in Myotonic Dystrophy Type 1Sara J Johnson, Hannah L Johnson, Reid T Powell, et al.
Molecular Cell|August 2, 2002
Loss of the muscle-specific chloride channel in type 1 myotonic dystrophy due to misregulated alternative splicingNicolas Charlet-B, Rajesh S Savkur, Gopal Singh, et al.
Pageof 11

Showing results (71-80 of 106) with videos related to

Sort By:
Pageof 11
The Journal of Clinical Investigation|September 8, 2007
Elevation of RNA-binding protein CUGBP1 is an early event in an inducible heart-specific mouse model of myotonic dystrophyGuey-Shin Wang, Debra L Kearney, Mariella De Biasi, et al.
Human Molecular Genetics|January 7, 2010
Heart-specific overexpression of CUGBP1 reproduces functional and molecular abnormalities of myotonic dystrophy type 1Misha Koshelev, Satyam Sarma, Roger E Price, et al.
Iscience|October 30, 2024
Alternatively spliced MAP4 isoforms have key roles in maintaining microtubule organization and skeletal muscle functionLathan Lucas, Larissa Nitschke, Brandon Nguyen, et al.
The International Journal of Biochemistry & Cell Biology|October 15, 2018
Modulation of alternative splicing of trafficking genes by genome editing reveals functional consequences in muscle biologyR Eric Blue, Amrita Koushik, Nichlas M Engels, et al.
Cell Reports|January 14, 2014
The Mef2 transcription network is disrupted in myotonic dystrophy heart tissue, dramatically altering miRNA and mRNA expressionAuinash Kalsotra, Ravi K Singh, Priyatansh Gurha, et al.
Proceedings of the National Academy of Sciences of the United States of America|February 15, 2008
Expanded CTG repeats within the DMPK 3' UTR causes severe skeletal muscle wasting in an inducible mouse model for myotonic dystrophyJames P Orengo, Pierre Chambon, Daniel Metzger, et al.
Human Molecular Genetics|August 30, 2007
Expression, localization and tau exon 10 splicing activity of the brain RNA-binding protein TNRC4J Paul Chapple, Karen Anthony, Teresa Rodriguez Martin, et al.
Elife|August 23, 2017
Extensive alternative splicing transitions during postnatal skeletal muscle development are required for calcium handling functionsAmy E Brinegar, Zheng Xia, James Anthony Loehr, et al.
Molecular and Cellular Biology|October 17, 2024
Small Molecule Screening Identifies HSP90 as a Modifier of RNA Foci in Myotonic Dystrophy Type 1Sara J Johnson, Hannah L Johnson, Reid T Powell, et al.
Molecular Cell|August 2, 2002
Loss of the muscle-specific chloride channel in type 1 myotonic dystrophy due to misregulated alternative splicingNicolas Charlet-B, Rajesh S Savkur, Gopal Singh, et al.
Pageof 11