Showing results (71-80 of 80) with videos related to

Sort By:
Pageof 8
You have reached the last page of results.This site can display upto 80 results.
Life Science Alliance|January 11, 2023
Functional substitutions of amino acids that differ between GDF11 and GDF8 impact skeletal development and skeletal muscleJohn Lian, Ryan G Walker, Andrea D'Amico, et al.
Proceedings of the National Academy of Sciences of the United States of America|July 19, 2019
Structural characterization of an activin class ternary receptor complex reveals a third paradigm for receptor specificityErich J Goebel, Richard A Corpina, Cynthia S Hinck, et al.
Plos One|June 4, 2019
The anti-sigma factor MucA of Pseudomonas aeruginosa: Dramatic differences of a mucA22 vs. a ΔmucA mutant in anaerobic acidified nitrite sensitivity of planktonic and biofilm bacteria in vitro and during chronic murine lung infectionWarunya Panmanee, Shengchang Su, Michael J Schurr, et al.
Scientific Reports|February 1, 2022
Visceral adipose tissue remodeling in pancreatic ductal adenocarcinoma cachexia: the role of activin A signalingPauline C Xu, Mikyoung You, Seok-Yeong Yu, et al.
Circulation Research|October 23, 2015
Circulating Growth Differentiation Factor 11/8 Levels Decline With AgeTommaso Poggioli, Ana Vujic, Peiguo Yang, et al.
BMC Biology|March 5, 2017
Structural basis for potency differences between GDF8 and GDF11Ryan G Walker, Magdalena Czepnik, Erich J Goebel, et al.
Elife|June 10, 2020
Activin A forms a non-signaling complex with ACVR1 and type II Activin/BMP receptors via its finger 2 tip loopSenem Aykul, Richard A Corpina, Erich J Goebel, et al.
Nature Structural & Molecular Biology|November 14, 2017
A consensus model of human apolipoprotein A-I in its monomeric and lipid-free stateJohn T Melchior, Ryan G Walker, Allison L Cooke, et al.
Nature Communications|July 15, 2025
Activated GDF11/8 subforms predict cardiovascular events and mortality in humansRyan G Walker, Tomohiro Kato, Laura Ben Driss, et al.
Human Mutation|June 20, 2019
Mutations in GDF11 and the extracellular antagonist, Follistatin, as a likely cause of Mendelian forms of orofacial clefting in humansTimothy C Cox, Andrew C Lidral, Jason C McCoy, et al.
Pageof 8