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Blood Advances|April 26, 2024
Myosin-1C augments endothelial secretion of von Willebrand factor by linking contractile actomyosin machinery to the plasma membraneSammy El-Mansi, Tom P Mitchell, Golzar Mobayen, et al.
The Journal of Cell Biology|August 17, 2011
Actomyosin II contractility expels von Willebrand factor from Weibel-Palade bodies during exocytosisThomas D Nightingale, Ian J White, Emily L Doyle, et al.
Plos Biology|December 3, 2019
Dynamic trafficking and turnover of JAM-C is essential for endothelial cell migrationKatja B Kostelnik, Amy Barker, Christopher Schultz, et al.
Cell Death & Disease|February 20, 2022
Genome wide CRISPR/Cas9 screen identifies the coagulation factor IX (F9) as a regulator of senescencePaula Carpintero-Fernández, Michela Borghesan, Olga Eleftheriadou, et al.
Blood|December 23, 2022
Proximity proteomics identifies septins and PAK2 as decisive regulators of actomyosin-mediated expulsion of von Willebrand factorSammy El-Mansi, Christopher L Robinson, Katja B Kostelnik, et al.
Science Signaling|May 23, 2023
Neuropilin-1 interacts with VE-cadherin and TGFBR2 to stabilize adherens junctions and prevent activation of endothelium under flowEmy Bosseboeuf, Anissa Chikh, Ahmed Bey Chaker, et al.
Circulation Research|November 4, 2025
TRIB3 Links Endoplasmic Reticulum Stress to Impaired Efferocytosis in AtherosclerosisAarushi Singhal, Stefan Russo, Umesh Kumar Dhawan, et al.
EMBO Reports|June 25, 2024
Senescent endothelial cells promote pathogenic neutrophil trafficking in inflamed tissuesLoïc Rolas, Monja Stein, Anna Barkaway, et al.
Immunity|August 7, 2021
Autophagy modulates endothelial junctions to restrain neutrophil diapedesis during inflammationNatalia Reglero-Real, Lorena Pérez-Gutiérrez, Azumi Yoshimura, et al.
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