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Thomas Dechat

Showing results (11-20 of 26) with videos related to

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Genome Research|January 23, 2016
A-type lamins bind both hetero- and euchromatin, the latter being regulated by lamina-associated polypeptide 2 alphaKevin Gesson, Philipp Rescheneder, Michael P Skoruppa, et al.
Journal of Cell Science|February 8, 2007
LAP2alpha-binding protein LINT-25 is a novel chromatin-associated protein involved in cell cycle exitNana Naetar, Sabine Hutter, Daniela Dorner, et al.
Genes & Development|April 3, 2008
Nuclear lamins: major factors in the structural organization and function of the nucleus and chromatinThomas Dechat, Katrin Pfleghaar, Kaushik Sengupta, et al.
The Journal of Clinical Investigation|July 10, 2009
Introducing intermediate filaments: from discovery to diseaseJohn E Eriksson, Thomas Dechat, Boris Grin, et al.
Journal of Virological Methods|October 3, 2017
Monolithic anion-exchange chromatography yields rhinovirus of high purityGünter Allmaier, Dieter Blaas, Christina Bliem, et al.
Journal of Cell Science|November 18, 2004
LAP2alpha and BAF transiently localize to telomeres and specific regions on chromatin during nuclear assemblyThomas Dechat, Andreas Gajewski, Barbara Korbei, et al.
Elife|February 19, 2021
LAP2alpha maintains a mobile and low assembly state of A-type lamins in the nuclear interiorNana Naetar, Konstantina Georgiou, Christian Knapp, et al.
PNAS Nexus|April 9, 2024
Twisted-plywood-like tissue formation <i>in vitro</i>. Does curvature do the twist?Barbara Schamberger, Sebastian Ehrig, Thomas Dechat, et al.
The Journal of Biological Chemistry|January 11, 2007
Lamina-associated polypeptide 2-alpha forms homo-trimers via its C terminus, and oligomerization is unaffected by a disease-causing mutationLuc Snyers, Sylvia Vlcek, Thomas Dechat, et al.
Journal of Cell Science|February 28, 2013
The muscle dystrophy-causing ΔK32 lamin A/C mutant does not impair the functions of the nucleoplasmic lamin-A/C-LAP2α complex in miceUrsula Pilat, Thomas Dechat, Anne T Bertrand, et al.
Pageof 3

Showing results (11-20 of 26) with videos related to

Sort By:
Pageof 3
Genome Research|January 23, 2016
A-type lamins bind both hetero- and euchromatin, the latter being regulated by lamina-associated polypeptide 2 alphaKevin Gesson, Philipp Rescheneder, Michael P Skoruppa, et al.
Journal of Cell Science|February 8, 2007
LAP2alpha-binding protein LINT-25 is a novel chromatin-associated protein involved in cell cycle exitNana Naetar, Sabine Hutter, Daniela Dorner, et al.
Genes & Development|April 3, 2008
Nuclear lamins: major factors in the structural organization and function of the nucleus and chromatinThomas Dechat, Katrin Pfleghaar, Kaushik Sengupta, et al.
The Journal of Clinical Investigation|July 10, 2009
Introducing intermediate filaments: from discovery to diseaseJohn E Eriksson, Thomas Dechat, Boris Grin, et al.
Journal of Virological Methods|October 3, 2017
Monolithic anion-exchange chromatography yields rhinovirus of high purityGünter Allmaier, Dieter Blaas, Christina Bliem, et al.
Journal of Cell Science|November 18, 2004
LAP2alpha and BAF transiently localize to telomeres and specific regions on chromatin during nuclear assemblyThomas Dechat, Andreas Gajewski, Barbara Korbei, et al.
Elife|February 19, 2021
LAP2alpha maintains a mobile and low assembly state of A-type lamins in the nuclear interiorNana Naetar, Konstantina Georgiou, Christian Knapp, et al.
PNAS Nexus|April 9, 2024
Twisted-plywood-like tissue formation <i>in vitro</i>. Does curvature do the twist?Barbara Schamberger, Sebastian Ehrig, Thomas Dechat, et al.
The Journal of Biological Chemistry|January 11, 2007
Lamina-associated polypeptide 2-alpha forms homo-trimers via its C terminus, and oligomerization is unaffected by a disease-causing mutationLuc Snyers, Sylvia Vlcek, Thomas Dechat, et al.
Journal of Cell Science|February 28, 2013
The muscle dystrophy-causing ΔK32 lamin A/C mutant does not impair the functions of the nucleoplasmic lamin-A/C-LAP2α complex in miceUrsula Pilat, Thomas Dechat, Anne T Bertrand, et al.
Pageof 3