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Cell Cycle (Georgetown, Tex.)|October 13, 2006
Activation of beta-catenin signaling pathways by classical G-protein-coupled receptors: mechanisms and consequences in cycling and non-cycling cellsSergey P Shevtsov, Syed Haq, Thomas ForceJournal of the American College of Cardiology|January 19, 2013
Cancer genetics and the cardiotoxicity of the therapeuticsHind Lal, Kyle L Kolaja, Thomas ForceJournal of the American College of Cardiology|February 15, 2005
Highlights of the 2004 scientific sessions of the Heart Failure Society of America, Toronto, Canada, September 12 to 15, 2004Peter Liu, Marvin A Konstam, Thomas ForceJournal of Molecular and Cellular Cardiology|December 18, 2010
Targeting GSK-3 family members in the heart: a very sharp double-edged swordHui Cheng, James Woodgett, Mia Maamari, et al.Circulation Journal : Official Journal of the Japanese Circulation Society|June 6, 2014
Troponin I-interacting protein kinase: a novel cardiac-specific kinase, emerging as a molecular target for the treatment of cardiac diseaseHind Lal, Firdos Ahmad, Shan Parikh, et al.Circulation Research|January 2, 2015
The GSK-3 family as therapeutic target for myocardial diseasesHind Lal, Firdos Ahmad, James Woodgett, et al.Journal of Cardiac Failure|January 30, 2003
Stretch-activated pathways and left ventricular remodelingThomas Force, Ashour Michael, Heiko Kilter, et al.Drug Discovery Today. Disease Mechanisms|February 1, 2011
Developing small molecules to inhibit kinases unkind to the heart: p38 MAPK as a case in pointMichael S Marber, Jeffery D Molkentin, Thomas ForceNature Reviews. Cancer|April 26, 2007
Molecular mechanisms of cardiotoxicity of tyrosine kinase inhibitionThomas Force, Daniela S Krause, Richard A Van EttenPhysiological Reviews|November 22, 2019
mRNA Metabolism in Cardiac Development and Disease: Life After TranscriptionChen Gao, Yibin WangPageof 55