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Blood|June 10, 2008
Differential effects of donor T-cell cytokines on outcome with continuous bortezomib administration after allogeneic bone marrow transplantationKai Sun, Minghui Li, Thomas J Sayers, et al.
Blood|January 11, 2003
Antigen-primed CD8+ T cells can mediate resistance, preventing allogeneic marrow engraftment in the simultaneous absence of perforin-, CD95L-, TNFR1-, and TRAIL-dependent killingMasanobu Komatsu, Michele Mammolenti, Monica Jones, et al.
Molecular Carcinogenesis|April 8, 2015
Impact of dietary components on NK and Treg cell function for cancer preventionYoung S Kim, Thomas J Sayers, Nancy H Colburn, et al.
Journal of Natural Products|November 12, 2010
Clerodane diterpenes from Casearia arguta that act as synergistic TRAIL sensitizersEmily L Whitson, Cheryl L Thomas, Curtis J Henrich, et al.
Scientific Reports|December 4, 2018
Sensitization of renal carcinoma cells to TRAIL-induced apoptosis by rocaglamide and analogsAncy D Nalli, Lauren E Brown, Cheryl L Thomas, et al.
Annals of the New York Academy of Sciences|December 31, 2005
The proteasome inhibitor bortezomib (Velcade) sensitizes some human tumor cells to Apo2L/TRAIL-mediated apoptosisAlan D Brooks, Teresa Ramirez, Uhi Toh, et al.
Cancer Research|August 6, 2009
Antigen presented by tumors in vivo determines the nature of CD8+ T-cell cytotoxicityAnil Shanker, Alan D Brooks, Kristen M Jacobsen, et al.
The Journal of Experimental Medicine|November 17, 2004
NKG2D recognition and perforin effector function mediate effective cytokine immunotherapy of cancerMark J Smyth, Jeremy Swann, Janice M Kelly, et al.
The Journal of Biological Chemistry|April 8, 2004
Discordant regulation of granzyme H and granzyme B expression in human lymphocytesKarin A Sedelies, Thomas J Sayers, Kirsten M Edwards, et al.
Molecular Cancer Therapeutics|June 3, 2010
Bortezomib sensitizes human esophageal squamous cell carcinoma cells to TRAIL-mediated apoptosis via activation of both extrinsic and intrinsic apoptosis pathwaysNaoko Seki, Uhi Toh, Thomas J Sayers, et al.
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