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Methods in Molecular Biology (Clifton, N.J.)|September 8, 2011
The role of heat shock factors in stress-induced transcriptionYue Zhang, Shiuh-Dih Chou, Ayesha Murshid, et al.
Biochemical Society Transactions|August 20, 2021
Extracellular Hsp90 and protection of neuronal cells through Nrf2Stuart K Calderwood, Thiago J Borges, Takanori Eguchi, et al.
Cells|April 26, 2020
HSF1: Primary Factor in Molecular Chaperone Expression and a Major Contributor to Cancer MorbidityThomas L Prince, Benjamin J Lang, Martin E Guerrero-Gimenez, et al.
Proceedings of the National Academy of Sciences of the United States of America|July 29, 2016
Gambogic acid identifies an isoform-specific druggable pocket in the middle domain of Hsp90βKendrick H Yim, Thomas L Prince, Shiwei Qu, et al.
Scientific Reports|May 5, 2018
HSP90 inhibitors disrupt a transient HSP90-HSF1 interaction and identify a noncanonical model of HSP90-mediated HSF1 regulationToshiki Kijima, Thomas L Prince, Megan L Tigue, et al.
Cancer Cell|January 29, 2013
Englerin A stimulates PKCθ to inhibit insulin signaling and to simultaneously activate HSF1: pharmacologically induced synthetic lethalityCarole Sourbier, Bradley T Scroggins, Ranjala Ratnayake, et al.
Plos One|October 31, 2015
Client Proteins and Small Molecule Inhibitors Display Distinct Binding Preferences for Constitutive and Stress-Induced HSP90 Isoforms and Their Conformationally Restricted MutantsThomas L Prince, Toshiki Kijima, Manabu Tatokoro, et al.
Cancer Letters|April 9, 2025
Chaperone directed heterobifunctional molecules circumvent KRASG12C inhibitor resistanceInes Pulido, Qiyue Luan, Sara Pastor-Puente, et al.
Molecular Cancer Therapeutics|October 23, 2025
Dual Inhibitors of KRASG12D and HSP90 are Effective Against KRASG12D Inhibitor ResistanceInés Pulido, Laura C Gunder, Chenghao Ying, et al.
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