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Journal of Neuropathology and Experimental Neurology|August 10, 2006
Synaptic vulnerability in neurodegenerative diseaseThomas M Wishart, Simon H Parson, Thomas H GillingwaterJournal of Anatomy|May 31, 2008
Rapid loss of motor nerve terminals following hypoxia-reperfusion injury occurs via mechanisms distinct from classic Wallerian degenerationBecki Baxter, Thomas H Gillingwater, Simon H ParsonPlos One|March 17, 2011
Induction of cell stress in neurons from transgenic mice expressing yellow fluorescent protein: implications for neurodegeneration researchLaura H Comley, Thomas M Wishart, Becki Baxter, et al.Anatomical Sciences Education|January 7, 2012
Development of a supported self-directed learning approach for anatomy educationGordon S Findlater, Fanney Kristmundsdottir, Simon H Parson, et al.Neuromuscular Disorders : NMD|July 28, 2016
Commonality amid diversity: Multi-study proteomic identification of conserved disease mechanisms in spinal muscular atrophyHeidi R Fuller, Thomas H Gillingwater, Thomas M WishartFASEB Journal : Official Publication of the Federation of American Societies for Experimental Biology|January 14, 2011
The response of neuromuscular junctions to injury is developmentally regulatedLyndsay M Murray, Laura H Comley, Thomas H Gillingwater, et al.Neuroscience Letters|April 16, 2013
Increasing SMN levels using the histone deacetylase inhibitor SAHA ameliorates defects in skeletal muscle microvasculature in a mouse model of severe spinal muscular atrophyEilidh Somers, Markus Riessland, Julia Schreml, et al.Journal of Anatomy|February 24, 2018
Developmental and degenerative cardiac defects in the Taiwanese mouse model of severe spinal muscular atrophyGillian K Maxwell, Eva Szunyogova, Hannah K Shorrock, et al.Plos One|December 3, 2010
Synaptic protection in the brain of WldS mice occurs independently of age but is sensitive to gene-doseAnn K Wright, Thomas M Wishart, Cali A Ingham, et al.Human Molecular Genetics|November 4, 2009
Pre-symptomatic development of lower motor neuron connectivity in a mouse model of severe spinal muscular atrophyLyndsay M Murray, Sheena Lee, Dirk Bäumer, et al.Pageof 22