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Biochimica Et Biophysica Acta|December 11, 2012
What can naturally occurring mutations tell us about Ca(v)1.x channel function?Thomas Stockner, Alexandra Koschak
Channels (Austin, Tex.)|December 15, 2010
Impact of gating modulation in CaV1.3 L-type calcium channelsAlexandra Koschak
Neuropharmacology|July 4, 2017
Relevance of tissue specific subunit expression in channelopathiesHartwig Seitter, Alexandra Koschak
Channels (Austin, Tex.)|March 25, 2025
Exploring the potential for gene therapy in Cav1.4-related retinal channelopathiesMatthias Ganglberger, Alexandra Koschak
Physiological Reviews|August 2, 2018
Voltage-Gated Calcium Channels: Key Players in Sensory Coding in the Retina and the Inner EarTina Pangrsic, Joshua H Singer, Alexandra Koschak
Pflugers Archiv : European Journal of Physiology|March 10, 2010
Channelopathies in Cav1.1, Cav1.3, and Cav1.4 voltage-gated L-type Ca2+ channelsJörg Striessnig, Hanno Jörn Bolz, Alexandra Koschak
The Journal of Pharmacology and Experimental Therapeutics|April 17, 2009
Use-dependent block of voltage-gated Cav2.1 Ca2+ channels by petasins and eudesmol isomersSilja Horak, Alexandra Koschak, Hermann Stuppner, et al.
Frontiers in Cellular Neuroscience|May 27, 2021
Investigating the Mechanism of Sodium Binding to SERT Using Direct SimulationsDániel Szöllősi, Thomas Stockner
Channels (Austin, Tex.)|August 15, 2015
Gain-of-function nature of Cav1.4 L-type calcium channels alters firing properties of mouse retinal ganglion cellsDagmar Knoflach, Klaus Schicker, Martin Glösmann, et al.
Cells|January 21, 2022
Sodium Binding Stabilizes the Outward-Open State of SERT by Limiting Bundle Domain MotionsDániel Szöllősi, Thomas Stockner
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