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The EMBO Journal|October 9, 2003
Monocytic cells hyperacetylate chromatin protein HMGB1 to redirect it towards secretionTiziana Bonaldi, Fabio Talamo, Paola Scaffidi, et al.Molecular and Cellular Biology|July 1, 2004
Global nature of dynamic protein-chromatin interactions in vivo: three-dimensional genome scanning and dynamic interaction networks of chromatin proteinsRobert D Phair, Paola Scaffidi, Cem Elbi, et al.Nucleic Acids Research|September 23, 2025
Mammalian H4K16ac regulates the spatiotemporal order of genome replication rather than gene expressionMarta Milan, Valeria Runfola, Manthan Patel, et al.Geroscience|June 9, 2025
Quantifying the impact of treatment delays on breast cancer survival outcomes: a comprehensive meta-analysisZoltan Ungvari, Mónika Fekete, Annamaria Buda, et al.International Journal of Molecular Sciences|October 6, 2020
Label-Free Mass Spectrometry-Based Quantification of Linker Histone H1 Variants in Clinical SamplesRoberta Noberini, Cristina Morales Torres, Evelyn Oliva Savoia, et al.Nature Structural & Molecular Biology|June 12, 2023
H4K16ac activates the transcription of transposable elements and contributes to their cis-regulatory functionDebosree Pal, Manthan Patel, Fanny Boulet, et al.EMBO Reports|July 24, 2004
HMGB1 is an endogenous immune adjuvant released by necrotic cellsPatrizia Rovere-Querini, Annalisa Capobianco, Paola Scaffidi, et al.Nature Communications|July 17, 2019
Patient-specific cancer genes contribute to recurrently perturbed pathways and establish therapeutic vulnerabilities in esophageal adenocarcinomaThanos P Mourikis, Lorena Benedetti, Elizabeth Foxall, et al.Cancer Cell|November 4, 2022
Selective advantage of epigenetically disrupted cancer cells via phenotypic inertiaIoannis Loukas, Fabrizio Simeoni, Marta Milan, et al.Science (New York, N.Y.)|October 7, 2016
The linker histone H1.0 generates epigenetic and functional intratumor heterogeneityCristina Morales Torres, Alva Biran, Matthew J Burney, et al.Pageof 4