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Scandinavian Journal of Gastroenterology|June 15, 2007
Gastroesophageal reflux disease leads to major alterations in lectin-binding in the columnar epithelium of the gastroesophageal junctionHelmut Neumann, Thomas Wex, Michael Vieth, et al.
Digestive Diseases and Sciences|July 30, 2010
H. pylori infection is a key risk factor for proximal gastric cancerJan Bornschein, Michael Selgrad, Maren Warnecke, et al.
BMC Gastroenterology|May 27, 2011
Mucosal Progranulin expression is induced by H. pylori, but independent of Secretory Leukocyte Protease Inhibitor (SLPI) expressionThomas Wex, Doerthe Kuester, Cornelius Schönberg, et al.
Helicobacter|January 21, 2006
Helicobacter pylori infection, but not low-dose aspirin, results in a local reduction of the secretory leukocyte protease inhibitor in gastroduodenal mucosaThomas Wex, Siying Ye, Gerhard Treiber, et al.
International Journal of Hematology|January 7, 2003
Plasma leptin and leptin receptor expression in childhood acute lymphoblastic leukemiaHeike Wex, Edita Ponelis, Thomas Wex, et al.
Helicobacter|November 9, 2011
Protease-activated receptor-2 (PAR2) in human gastric mucosa as mediator of proinflammatory effects in Helicobacter pylori infectionArne Kandulski, Doerthe Kuester, Klaus Mönkemüller, et al.
Peptides|May 26, 2009
Zonulin is not increased in the cardiac and esophageal mucosa of patients with gastroesophageal reflux diseaseThomas Wex, Klaus Mönkemüller, Doerthe Kuester, et al.
Infection and Immunity|March 25, 2004
Helicobacter pylori-mediated gastritis induces local downregulation of secretory leukocyte protease inhibitor in the antrumThomas Wex, Gerhard Treiber, Manfred Nilius, et al.
Cancer Letters|June 24, 2008
The long-term effect of Helicobacter pylori eradication on COX-1/2, 5-LOX and leukotriene receptors in patients with a risk gastritis phenotype--a link to gastric carcinogenesisMarino Venerito, Doerthe Kuester, Thomas Wex, et al.
Biochemical and Biophysical Research Communications|September 11, 2004
Identification of a novel isoform predominantly expressed in gastric tissue and a triple-base pair polymorphism of the cathepsin W geneChristian Meinhardt, Ulrich Peitz, Gerhard Treiber, et al.
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