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Journal of the American Society of Nephrology : JASN|April 23, 2003
Glucose, glycation, and RAGE: implications for amplification of cellular dysfunction in diabetic nephropathyThoralf Wendt, Nozomu Tanji, Jiancheng Guo, et al.
Diabetes|February 28, 2003
Acute hyperglycemia causes intracellular formation of CML and activation of ras, p42/44 MAPK, and nuclear factor kappaB in PBMCsStephan Schiekofer, Martin Andrassy, Jiang Chen, et al.
Diabetes & Vascular Disease Research|November 25, 2005
RAGE and its ligands: a lasting memory in diabetic complications?Shi-Fang Yan, Ravichandran Ramasamy, Loredana G Bucciarelli, et al.
Circulation|November 27, 2002
RAGE blockade stabilizes established atherosclerosis in diabetic apolipoprotein E-null miceLoredana G Bucciarelli, Thoralf Wendt, Wu Qu, et al.
Proceedings of the National Academy of Sciences of the United States of America|February 13, 2003
A mechanism converting psychosocial stress into mononuclear cell activationAngelika Bierhaus, Jutta Wolf, Martin Andrassy, et al.
Archives of Biochemistry and Biophysics|October 22, 2003
Blockade of receptor for advanced glycation endproducts: a new target for therapeutic intervention in diabetic complications and inflammatory disordersBarry I Hudson, Loredana G Bucciarelli, Thoralf Wendt, et al.
Investigative Ophthalmology & Visual Science|July 27, 2005
The RAGE axis in early diabetic retinopathyGaetano R Barile, Sophia I Pachydaki, Samir R Tari, et al.
FASEB Journal : Official Publication of the Federation of American Societies for Experimental Biology|December 4, 2004
RAGE modulates peripheral nerve regeneration via recruitment of both inflammatory and axonal outgrowth pathwaysLing Ling Rong, Shi-Fang Yan, Thoralf Wendt, et al.
The American Journal of Pathology|September 28, 2006
Posttranslationally modified proteins as mediators of sustained intestinal inflammationMartin Andrassy, John Igwe, Frank Autschbach, et al.
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