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American Journal of Physiology. Heart and Circulatory Physiology|July 18, 2006
Low-dose simvastatin improves survival and ventricular function via eNOS in congestive heart failureJames J M Greer, Aman K Kakkar, John W Elrod, et al.
The Biochemical Journal|July 15, 2017
Integration of flux measurements to resolve changes in anabolic and catabolic metabolism in cardiac myocytesAndrew A Gibb, Pawel K Lorkiewicz, Yu-Ting Zheng, et al.
American Journal of Physiology. Heart and Circulatory Physiology|March 13, 2012
O-GlcNAc signaling is essential for NFAT-mediated transcriptional reprogramming during cardiomyocyte hypertrophyHeberty T Facundo, Robert E Brainard, Lewis J Watson, et al.
Disease Models & Mechanisms|July 14, 2019
RDH10 function is necessary for spontaneous fetal mouth movement that facilitates palate shelf elevationRegina M Friedl, Swetha Raja, Melissa A Metzler, et al.
The Journal of Biological Chemistry|September 4, 2014
MicroRNA-539 is up-regulated in failing heart, and suppresses O-GlcNAcase expressionSenthilkumar Muthusamy, Angelica M DeMartino, Lewis J Watson, et al.
Primary Dental Care : Journal of the Faculty of General Dental Practitioners (UK)|March 21, 2002
Advances in orthodonticsSusan J Cunningham, Steven P Jones, Samantha J Hodges, et al.
Circulation|February 21, 2008
Cardioprotection by N-acetylglucosamine linkage to cellular proteinsSteven P Jones, Natasha E Zachara, Gladys A Ngoh, et al.
Circulation|December 22, 2004
Functional integration of electrically active cardiac derivatives from genetically engineered human embryonic stem cells with quiescent recipient ventricular cardiomyocytes: insights into the development of cell-based pacemakersTian Xue, Hee Cheol Cho, Fadi G Akar, et al.
Cell Metabolism|February 12, 2013
O-GlcNAc signaling entrains the circadian clock by inhibiting BMAL1/CLOCK ubiquitinationMin-Dian Li, Hai-Bin Ruan, Michael E Hughes, et al.
FASEB Journal : Official Publication of the Federation of American Societies for Experimental Biology|February 27, 2025
Neutrophil-secreted CHI3L1 exacerbates cardiac dysfunction and inflammation after myocardial infarctionJonah K Stephan, Taylor Knerr, Zhen Gu, et al.
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