Showing results (131-140 of 151) with videos related to
Sort By:
Pageof 16
American Journal of Physiology. Heart and Circulatory Physiology|September 13, 2003
Endothelial nitric oxide synthase overexpression attenuates myocardial reperfusion injurySteven P Jones, James J M Greer, Aman K Kakkar, et al.American Journal of Physiology. Heart and Circulatory Physiology|May 27, 2022
Metabolic signatures of pregnancy-induced cardiac growthKyle L Fulghum, Juliette B Smith, Julia Chariker, et al.Plos One|December 25, 2013
High fat feeding in mice is insufficient to induce cardiac dysfunction and does not exacerbate heart failureRobert E Brainard, Lewis J Watson, Angelica M Demartino, et al.Journal of Molecular and Cellular Cardiology|November 30, 2024
Hyaluronan provokes inflammation but suppresses phagocytotic function in macrophagesTimothy N Audam, Caitlin M Howard, Danielle T Little, et al.Journal of Molecular and Cellular Cardiology|April 9, 2018
Deficiency of aldose reductase exacerbates early pressure overload-induced cardiac dysfunction and autophagy in miceShahid P Baba, Deqing Zhang, Mahavir Singh, et al.Redox Biology|June 10, 2018
Cardiac-specific overexpression of aldehyde dehydrogenase 2 exacerbates cardiac remodeling in response to pressure overloadSujith Dassanayaka, Yuting Zheng, Andrew A Gibb, et al.The Journal of Pharmacology and Experimental Therapeutics|March 11, 2010
TRO40303, a new cardioprotective compound, inhibits mitochondrial permeability transitionSophie Schaller, Stéphanie Paradis, Gladys A Ngoh, et al.American Journal of Physiology. Cell Physiology|January 28, 2025
Activated cardiac fibroblasts are a primary source of high-molecular-weight hyaluronan productionDanielle T Little, Caitlin M Howard, Emma Pendergraft, et al.Basic Research in Cardiology|June 2, 2019
E2f1 deletion attenuates infarct-induced ventricular remodeling without affecting O-GlcNAcylationSujith Dassanayaka, Kenneth R Brittian, Andrea Jurkovic, et al.American Journal of Physiology. Heart and Circulatory Physiology|May 23, 2020
Cardiac mesenchymal cells from failing and nonfailing hearts limit ventricular dilation when administered late after infarctionTimothy N Audam, Yibing Nong, Alex Tomlin, et al.Pageof 16