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Circulation|September 2, 2017
Exercise-Induced Changes in Glucose Metabolism Promote Physiological Cardiac GrowthAndrew A Gibb, Paul N Epstein, Shizuka Uchida, et al.
Basic Research in Cardiology|March 17, 2017
Cardiomyocyte Ogt limits ventricular dysfunction in mice following pressure overload without affecting hypertrophySujith Dassanayaka, Robert E Brainard, Lewis J Watson, et al.
Plos One|November 30, 2020
Cardiomyocyte Oga haploinsufficiency increases O-GlcNAcylation but hastens ventricular dysfunction following myocardial infarctionSujith Dassanayaka, Kenneth R Brittian, Bethany W Long, et al.
Circulation. Heart Failure|April 26, 2014
Metabolomic analysis of pressure-overloaded and infarcted mouse heartsBrian E Sansbury, Angelica M DeMartino, Zhengzhi Xie, et al.
American Journal of Physiology. Heart and Circulatory Physiology|September 17, 2021
Cardiac PANK1 deletion exacerbates ventricular dysfunction during pressure overloadTimothy N Audam, Caitlin M Howard, Lauren F Garrett, et al.
Nature Communications|May 26, 2026
TAK1 drives inflammatory fibroblast acquisition and shapes myocardial infarction responses in male miceDaniel C Nguyen, Jonah K Stephan, Lianay Gutierrez Luque, et al.
Journal of Molecular and Cellular Cardiology|July 12, 2025
Pyruvate kinase splice variants in fibroblasts influence cardiac remodeling after myocardial infarction in male miceCollin K Wells, Daniel C Nguyen, Robert E Brainard, et al.
American Journal of Physiology. Heart and Circulatory Physiology|January 20, 2018
Guidelines for experimental models of myocardial ischemia and infarctionMerry L Lindsey, Roberto Bolli, John M Canty, et al.
American Journal of Physiology. Heart and Circulatory Physiology|October 8, 2021
Guidelines for in vivo mouse models of myocardial infarctionMerry L Lindsey, Keith R Brunt, Jonathan A Kirk, et al.
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