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The Journal of Cell Biology|November 8, 2006
Rapidly inducible changes in phosphatidylinositol 4,5-bisphosphate levels influence multiple regulatory functions of the lipid in intact living cellsPeter Varnai, Baskaran Thyagarajan, Tibor Rohacs, et al.
The Journal of Biological Chemistry|January 10, 2013
Interplay between calmodulin and phosphatidylinositol 4,5-bisphosphate in Ca2+-induced inactivation of transient receptor potential vanilloid 6 channelsChike Cao, Eleonora Zakharian, Istvan Borbiro, et al.
Methods in Enzymology|June 1, 2021
Methods to study phosphoinositide regulation of ion channelsYevgen Yudin, Luyu Liu, Janhavi Nagwekar, et al.
The Journal of Physiology|October 19, 2011
Decrease in phosphatidylinositol 4,5-bisphosphate levels mediates desensitization of the cold sensor TRPM8 channelsYevgen Yudin, Viktor Lukacs, Chike Cao, et al.
The Journal of Physiology|April 11, 2016
Phospholipase C δ4 regulates cold sensitivity in miceYevgen Yudin, Brianna Lutz, Yuan-Xiang Tao, et al.
Molecular Pharmacology|December 17, 2008
Phospholipase C-mediated regulation of transient receptor potential vanilloid 6 channels: implications in active intestinal Ca2+ transportBaskaran Thyagarajan, Bryan S Benn, Sylvia Christakos, et al.
The Journal of General Physiology|July 12, 2022
TMEM120A/TACAN inhibits mechanically activated PIEZO2 channelsJohn Smith Del Rosario, Matthew Gabrielle, Yevgen Yudin, et al.
Nature Communications|August 15, 2024
Phosphatidic acid is an endogenous negative regulator of PIEZO2 channels and mechanical sensitivityMatthew Gabrielle, Yevgen Yudin, Yujue Wang, et al.
Biorxiv : the Preprint Server for Biology|March 11, 2024
Phosphatidic acid is an endogenous negative regulator of PIEZO2 channels and mechanical sensitivityMatthew Gabrielle, Yevgen Yudin, Yujue Wang, et al.
The Journal of Biological Chemistry|October 1, 2022
Computational and functional studies of the PI(4,5)P2 binding site of the TRPM3 ion channel reveal interactions with other regulatorsSiyuan Zhao, Vincenzo Carnevale, Matthew Gabrielle, et al.
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