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The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|July 31, 2015
C-Terminally Truncated Forms of Tau, But Not Full-Length Tau or Its C-Terminal Fragments, Are Released from Neurons Independently of Cell DeathDaniel Kanmert, Adam Cantlon, Christina R Muratore, et al.
Alzheimer'S & Dementia : the Journal of the Alzheimer'S Association|April 8, 2015
The aqueous phase of Alzheimer's disease brain contains assemblies built from ∼4 and ∼7 kDa Aβ speciesJessica M Mc Donald, Tiernan T O'Malley, Wen Liu, et al.
The Biochemical Journal|May 3, 2014
Aβ dimers differ from monomers in structural propensity, aggregation paths and population of synaptotoxic assembliesTiernan T O'Malley, Nur Alia Oktaviani, Dainan Zhang, et al.
Brain : a Journal of Neurology|April 30, 2019
Identification of neurotoxic cross-linked amyloid-β dimers in the Alzheimer's brainGunnar Brinkmalm, Wei Hong, Zemin Wang, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|July 25, 2023
Tau and Amyloid β Protein in Patient-Derived Aqueous Brain Extracts Act Concomitantly to Disrupt Long-Term Potentiation in VivoTomas Ondrejcak, Igor Klyubin, Neng-Wei Hu, et al.
Nature Communications|July 12, 2018
An in vitro paradigm to assess potential anti-Aβ antibodies for Alzheimer's diseaseMing Jin, Brian O'Nuallain, Wei Hong, et al.
The New England Journal of Medicine|December 8, 2025
Prime Editing for p47phox-Deficient Chronic Granulomatous DiseaseJennifer L Gori, Elie Haddad, Haydar Frangoul, et al.
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