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Tiffany J Petrisko

Showing results (1-10 of 13) with videos related to

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Nature Reviews. Neurology|March 25, 2025
Knowing the enemy: strategic targeting of complement to treat Alzheimer diseaseAndrea J Tenner, Tiffany J Petrisko
Metabolic Brain Disease|August 18, 2021
Peripheral viral challenge increases c-fos level in cerebral neuronsTiffany J Petrisko, Gregory W Konat
Metabolic Brain Disease|January 15, 2019
Peripheral viral challenge exacerbates experimental autoimmune encephalomyelitisTiffany J Petrisko, Gregory W Konat
Advances in Immunology|November 30, 2021
Complement as a powerful "influencer" in the brain during development, adulthood and neurological disordersTiffany J Petrisko, Angela Gomez-Arboledas, Andrea J Tenner
Alzheimer'S & Dementia : the Journal of the Alzheimer'S Association|December 24, 2025
Basic Science and PathogenesisTiffany J Petrisko, Angela Gomez-Arboledas, Shu-Hui Chu, et al.
Biorxiv : the Preprint Server for Biology|September 26, 2025
Young adult microglial deletion of C1q reduces engulfment of synapses and prevents cognitive impairment in an aggressive Alzheimer's disease mouse modelTiffany J Petrisko, Shu-Hui Chu, Angela Gomez-Arboledas, et al.
Journal of Neuroinflammation|September 19, 2023
Influence of complement protein C1q or complement receptor C5aR1 on gut microbiota composition in wildtype and Alzheimer's mouse modelsTiffany J Petrisko, Matthew Gargus, Shu-Hui Chu, et al.
Journal of Neurochemistry|May 12, 2016
Peripherally restricted viral challenge elevates extracellular glutamate and enhances synaptic transmission in the hippocampusHolly C Hunsberger, Desheng Wang, Tiffany J Petrisko, et al.
Nature Communications|August 15, 2024
C5aR1 antagonism suppresses inflammatory glial responses and alters cellular signaling in an Alzheimer's disease mouse modelNicole D Schartz, Heidi Y Liang, Klebea Carvalho, et al.
Biorxiv : the Preprint Server for Biology|September 4, 2023
C5aR1 antagonism suppresses inflammatory glial gene expression and alters cellular signaling in an aggressive Alzheimer's modelNicole D Schartz, Heidi Y Liang, Klebea Carvalho, et al.
Pageof 2

Showing results (1-10 of 13) with videos related to

Sort By:
Pageof 2
Nature Reviews. Neurology|March 25, 2025
Knowing the enemy: strategic targeting of complement to treat Alzheimer diseaseAndrea J Tenner, Tiffany J Petrisko
Metabolic Brain Disease|August 18, 2021
Peripheral viral challenge increases c-fos level in cerebral neuronsTiffany J Petrisko, Gregory W Konat
Metabolic Brain Disease|January 15, 2019
Peripheral viral challenge exacerbates experimental autoimmune encephalomyelitisTiffany J Petrisko, Gregory W Konat
Advances in Immunology|November 30, 2021
Complement as a powerful "influencer" in the brain during development, adulthood and neurological disordersTiffany J Petrisko, Angela Gomez-Arboledas, Andrea J Tenner
Alzheimer'S & Dementia : the Journal of the Alzheimer'S Association|December 24, 2025
Basic Science and PathogenesisTiffany J Petrisko, Angela Gomez-Arboledas, Shu-Hui Chu, et al.
Biorxiv : the Preprint Server for Biology|September 26, 2025
Young adult microglial deletion of C1q reduces engulfment of synapses and prevents cognitive impairment in an aggressive Alzheimer's disease mouse modelTiffany J Petrisko, Shu-Hui Chu, Angela Gomez-Arboledas, et al.
Journal of Neuroinflammation|September 19, 2023
Influence of complement protein C1q or complement receptor C5aR1 on gut microbiota composition in wildtype and Alzheimer's mouse modelsTiffany J Petrisko, Matthew Gargus, Shu-Hui Chu, et al.
Journal of Neurochemistry|May 12, 2016
Peripherally restricted viral challenge elevates extracellular glutamate and enhances synaptic transmission in the hippocampusHolly C Hunsberger, Desheng Wang, Tiffany J Petrisko, et al.
Nature Communications|August 15, 2024
C5aR1 antagonism suppresses inflammatory glial responses and alters cellular signaling in an Alzheimer's disease mouse modelNicole D Schartz, Heidi Y Liang, Klebea Carvalho, et al.
Biorxiv : the Preprint Server for Biology|September 4, 2023
C5aR1 antagonism suppresses inflammatory glial gene expression and alters cellular signaling in an aggressive Alzheimer's modelNicole D Schartz, Heidi Y Liang, Klebea Carvalho, et al.
Pageof 2