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Biorxiv : the Preprint Server for Biology|July 9, 2024
Replicative senescence is ATM driven, reversible, and accelerated by hyperactivation of ATM at normoxiaAlexander Stuart, Titia de LangeAnnual Review of Biochemistry|June 11, 2004
Regulation of telomerase by telomeric proteinsAgata Smogorzewska, Titia de LangeThe Journal of Biological Chemistry|August 8, 2018
Protection of telomeres 1 proteins POT1a and POT1b can repress ATR signaling by RPA exclusion, but binding to CST limits ATR repression by POT1bKatja Kratz, Titia de LangeMolecular Cell|November 13, 2010
A Shld1-controlled POT1a provides support for repression of ATR signaling at telomeres through RPA exclusionYi Gong, Titia de LangeAnnual Review of Cell and Developmental Biology|August 2, 2011
The causes and consequences of polyploidy in normal development and cancerTeresa Davoli, Titia de LangeThe Journal of Biological Chemistry|June 15, 2007
The role of the poly(ADP-ribose) polymerase tankyrase1 in telomere length control by the TRF1 component of the shelterin complexJill R Donigian, Titia de LangeCurrent Biology : CB|May 30, 2006
Apollo, an Artemis-related nuclease, interacts with TRF2 and protects human telomeres in S phaseMegan van Overbeek, Titia de LangeNature Cell Biology|June 22, 2005
DNA processing is not required for ATM-mediated telomere damage response after TRF2 deletionGiulia B Celli, Titia de LangeNature|August 10, 2007
Protection of telomeres through independent control of ATM and ATR by TRF2 and POT1Eros Lazzerini Denchi, Titia de LangePageof 12