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Tobias Moll

Showing results (1-10 of 26) with videos related to

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Brain : a Journal of Neurology|November 15, 2019
Disrupted glycosylation of lipids and proteins is a cause of neurodegenerationTobias Moll, Pamela J Shaw, Johnathan Cooper-Knock
International Review of Neurobiology|May 27, 2024
Non-coding genome contribution to ALSTobias Moll, Calum Harvey, Elham Alhathli, et al.
Essays in Biochemistry|October 8, 2021
Membrane lipid raft homeostasis is directly linked to neurodegenerationTobias Moll, Jack N G Marshall, Nikita Soni, et al.
Current Opinion in Neurology|August 3, 2021
Advances in the genetic classification of amyotrophic lateral sclerosisJohnathan Cooper-Knock, Calum Harvey, Sai Zhang, et al.
Amyotrophic Lateral Sclerosis & Frontotemporal Degeneration|July 4, 2020
Concurrent sodium channelopathies and amyotrophic lateral sclerosis supports shared pathogenesisJohn P Franklin, Johnathan Cooper-Knock, Aravindhan Baheerathan, et al.
Cell Reports|June 28, 2024
Stress-induced TDP-43 nuclear condensation causes splicing loss of function and STMN2 depletionWan-Ping Huang, Brittany C S Ellis, Rachel E Hodgson, et al.
Ebiomedicine|May 29, 2021
Physical exercise is a risk factor for amyotrophic lateral sclerosis: Convergent evidence from Mendelian randomisation, transcriptomics and risk genotypesThomas H Julian, Nicholas Glascow, A Dylan Fisher Barry, et al.
Brain : a Journal of Neurology|November 18, 2021
A review of Mendelian randomization in amyotrophic lateral sclerosisThomas H Julian, Sarah Boddy, Mahjabin Islam, et al.
Cell Reports|July 7, 2026
Striatal neuron dysfunction in C9ORF72-FTD/ALS is driven by AIS and potassium channel dysregulationIris-Stefania Pasniceanu, Manpreet S Atwal, Cleide Dos Santos Souza, et al.
Amyotrophic Lateral Sclerosis & Frontotemporal Degeneration|December 7, 2020
Simultaneous ALS and SCA2 associated with an intermediate-length <i>ATXN2</i> CAG-repeat expansionHelia Ghahremani Nezhad, John P Franklin, James J P Alix, et al.
Pageof 3

Showing results (1-10 of 26) with videos related to

Sort By:
Pageof 3
Brain : a Journal of Neurology|November 15, 2019
Disrupted glycosylation of lipids and proteins is a cause of neurodegenerationTobias Moll, Pamela J Shaw, Johnathan Cooper-Knock
International Review of Neurobiology|May 27, 2024
Non-coding genome contribution to ALSTobias Moll, Calum Harvey, Elham Alhathli, et al.
Essays in Biochemistry|October 8, 2021
Membrane lipid raft homeostasis is directly linked to neurodegenerationTobias Moll, Jack N G Marshall, Nikita Soni, et al.
Current Opinion in Neurology|August 3, 2021
Advances in the genetic classification of amyotrophic lateral sclerosisJohnathan Cooper-Knock, Calum Harvey, Sai Zhang, et al.
Amyotrophic Lateral Sclerosis & Frontotemporal Degeneration|July 4, 2020
Concurrent sodium channelopathies and amyotrophic lateral sclerosis supports shared pathogenesisJohn P Franklin, Johnathan Cooper-Knock, Aravindhan Baheerathan, et al.
Cell Reports|June 28, 2024
Stress-induced TDP-43 nuclear condensation causes splicing loss of function and STMN2 depletionWan-Ping Huang, Brittany C S Ellis, Rachel E Hodgson, et al.
Ebiomedicine|May 29, 2021
Physical exercise is a risk factor for amyotrophic lateral sclerosis: Convergent evidence from Mendelian randomisation, transcriptomics and risk genotypesThomas H Julian, Nicholas Glascow, A Dylan Fisher Barry, et al.
Brain : a Journal of Neurology|November 18, 2021
A review of Mendelian randomization in amyotrophic lateral sclerosisThomas H Julian, Sarah Boddy, Mahjabin Islam, et al.
Cell Reports|July 7, 2026
Striatal neuron dysfunction in C9ORF72-FTD/ALS is driven by AIS and potassium channel dysregulationIris-Stefania Pasniceanu, Manpreet S Atwal, Cleide Dos Santos Souza, et al.
Amyotrophic Lateral Sclerosis & Frontotemporal Degeneration|December 7, 2020
Simultaneous ALS and SCA2 associated with an intermediate-length <i>ATXN2</i> CAG-repeat expansionHelia Ghahremani Nezhad, John P Franklin, James J P Alix, et al.
Pageof 3