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Cancer Discovery|January 10, 2015
JAK-STAT pathway activation in malignant and nonmalignant cells contributes to MPN pathogenesis and therapeutic responseMaria Kleppe, Minsuk Kwak, Priya Koppikar, et al.Cancer Cell|August 18, 2012
ASXL1 mutations promote myeloid transformation through loss of PRC2-mediated gene repressionOmar Abdel-Wahab, Mazhar Adli, Lindsay M LaFave, et al.Elife|November 18, 2015
Cross-talk between PRMT1-mediated methylation and ubiquitylation on RBM15 controls RNA splicingLi Zhang, Ngoc-Tung Tran, Hairui Su, et al.Proceedings of the National Academy of Sciences of the United States of America|December 18, 2014
Genomic and functional analysis of leukemic transformation of myeloproliferative neoplasmsRaajit Rampal, Jihae Ahn, Omar Abdel-Wahab, et al.Cancer Cell|April 16, 2015
Mutational cooperativity linked to combinatorial epigenetic gain of function in acute myeloid leukemiaAlan H Shih, Yanwen Jiang, Cem Meydan, et al.The Journal of Experimental Medicine|November 13, 2013
Deletion of Asxl1 results in myelodysplasia and severe developmental defects in vivoOmar Abdel-Wahab, Jie Gao, Mazhar Adli, et al.Cell Reports|December 9, 2014
DNA hydroxymethylation profiling reveals that WT1 mutations result in loss of TET2 function in acute myeloid leukemiaRaajit Rampal, Altuna Alkalin, Jozef Madzo, et al.Nature Medicine|October 6, 2015
Loss of BAP1 function leads to EZH2-dependent transformationLindsay M LaFave, Wendy Béguelin, Richard Koche, et al.Nature Medicine|June 21, 2016
Distinct evolution and dynamics of epigenetic and genetic heterogeneity in acute myeloid leukemiaSheng Li, Francine E Garrett-Bakelman, Stephen S Chung, et al.Pageof 2