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Cancer Research|August 23, 2013
Activation of MAPK pathways due to DUSP4 loss promotes cancer stem cell-like phenotypes in basal-like breast cancerJustin M Balko, Luis J Schwarz, Neil E Bhola, et al.
British Journal of Clinical Pharmacology|September 27, 2022
Tumour, whole-blood, plasma and tissue concentrations of metformin in lung cancer patientsJoseph D Phillips, Darcy B Pooler, Dylan B Ness, et al.
Biorxiv : the Preprint Server for Biology|August 8, 2025
Endocrine therapy induces oxidative stress in ER+ breast cancer that sensitizes persister cells to ferroptosisSteven Tau, Malone Friedman, Alyssa M Roberts, et al.
Scientific Reports|December 6, 2018
Computational affinity maturation of camelid single-domain intrabodies against the nonamyloid component of alpha-synucleinSai Pooja Mahajan, Bunyarit Meksiriporn, Dujduan Waraho-Zhmayev, et al.
Experimental and Molecular Pathology|April 21, 2016
Targeted next-generation sequencing detects a high frequency of potentially actionable mutations in metastatic breast cancersKristen E Muller, Jonathan D Marotti, Francine B de Abreu, et al.
Molecular Oncology|June 11, 2019
Estrogen therapy induces an unfolded protein response to drive cell death in ER+ breast cancerSarah R Hosford, Kevin Shee, Jason D Wells, et al.
Marine Pollution Bulletin|March 15, 2011
Hypoxia in Manila Bay, Philippines during the northeast monsoonGil S Jacinto, Lara Patricia A Sotto, Maria Isabel S Senal, et al.
Proceedings of the National Academy of Sciences of the United States of America|December 16, 2004
Potent inhibition of huntingtin aggregation and cytotoxicity by a disulfide bond-free single-domain intracellular antibodyDavid W Colby, Yijia Chu, John P Cassady, et al.
Molecular Cancer Therapeutics|August 11, 2012
Discordant cellular response to presurgical letrozole in bilateral synchronous ER+ breast cancers with a KRAS mutation or FGFR1 gene amplificationJustin M Balko, Ingrid A Mayer, Melinda E Sanders, et al.
Journal of Molecular Biology|April 12, 2015
Structure of a single-chain Fv bound to the 17 N-terminal residues of huntingtin provides insights into pathogenic amyloid formation and suppressionErwin De Genst, Dimitri Y Chirgadze, Fabrice A C Klein, et al.
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