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Journal of the American College of Cardiology|April 14, 2018
Interplay Between Genetic Substrate, QTc Duration, and Arrhythmia Risk in Patients With Long QT SyndromeAndrea Mazzanti, Riccardo Maragna, Gaetano Vacanti, et al.JACC. Basic to Translational Science|January 23, 2018
Suppression of Arrhythmia by Enhancing Mitochondrial Ca2+ Uptake in Catecholaminergic Ventricular Tachycardia ModelsMaria K Schweitzer, Fabiola Wilting, Simon Sedej, et al.European Heart Journal|July 13, 2022
TRPM4 inhibition by meclofenamate suppresses Ca2+-dependent triggered arrhythmiasFrone Vandewiele, Andy Pironet, Griet Jacobs, et al.Heart Rhythm|June 7, 2008
Risk of death in the long QT syndrome when a sibling has diedElizabeth S Kaufman, Scott McNitt, Arthur J Moss, et al.Journal of the American Heart Association|May 30, 2020
Tetrodotoxin-Sensitive Neuronal-Type Na+ Channels: A Novel and Druggable Target for Prevention of Atrial FibrillationMark A Munger, Yusuf Olğar, Megan L Koleske, et al.Cardiovascular Research|December 25, 2014
Neuronal Na+ channel blockade suppresses arrhythmogenic diastolic Ca2+ releasePrzemysław B Radwański, Lucia Brunello, Rengasayee Veeraraghavan, et al.Circulation|June 4, 2014
Single delivery of an adeno-associated viral construct to transfer the CASQ2 gene to knock-in mice affected by catecholaminergic polymorphic ventricular tachycardia is able to cure the disease from birth to advanced ageMarco Denegri, Rossana Bongianino, Francesco Lodola, et al.Cell|July 3, 2003
FKBP12.6 deficiency and defective calcium release channel (ryanodine receptor) function linked to exercise-induced sudden cardiac deathXander H T Wehrens, Stephan E Lehnart, Fannie Huang, et al.JACC. Basic to Translational Science|October 18, 2016
Neuronal Na+ Channels Are Integral Components of Pro-arrhythmic Na+/Ca2+ Signaling Nanodomain That Promotes Cardiac Arrhythmias During β-adrenergic StimulationPrzemysław B Radwański, Hsiang-Ting Ho, Rengasayee Veeraraghavan, et al.Circulation|May 7, 2020
Desmoplakin Cardiomyopathy, a Fibrotic and Inflammatory Form of Cardiomyopathy Distinct From Typical Dilated or Arrhythmogenic Right Ventricular CardiomyopathyEric D Smith, Neal K Lakdawala, Nikolaos Papoutsidakis, et al.Pageof 24