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Oxidative Medicine and Cellular Longevity|January 3, 2022
Calcium-/Calmodulin-Dependent Protein Kinase II (CaMKII) Inhibition Induces Learning and Memory Impairment and ApoptosisJialu Wang, Xiaoxue Xu, Wanying Jia, et al.International Journal of Molecular Sciences|August 26, 2018
The Effect of Ca2+, Lobe-Specificity, and CaMKII on CaM Binding to NaV1.1Jianing Li, Zhiyi Yu, Jianjun Xu, et al.Scientific Reports|July 25, 2019
MTP18 is a Novel Regulator of Mitochondrial Fission in CNS Neuron Development, Axonal Growth, and Injury ResponsesAlexander Kreymerman, David N Buickians, Michael M Nahmou, et al.Epilepsia|March 22, 2025
Increased NaV1.2 expression and its interaction with CaM contribute to the hyperexcitability induced by prolonged inhibition of CaMKIIHongyue Liang, Ling Qin, Rui Feng, et al.Journal of Neuroinflammation|December 1, 2024
TRPM7 contributes to pyroptosis and its involvement in status epilepticusXin Tong, Yu Tong, Jiahe Zheng, et al.Pageof 6