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Neuroscience Letters|August 17, 2002
Reduction of neurotransmitter release by the exogenous H3 domain peptide of HPC-1/syntaxin 1A in cultured rat hippocampal neuronsTatsuya Mishima, Tomonori Fujiwara, Kimio AkagawaBehavioural Brain Research|July 5, 2021
Disturbance of the reciprocal-interaction between the OXTergic and DAergic systems in the CNS causes atypical social behavior in syntaxin 1A knockout miceTomonori Fujiwara, Takefumi Kofuji, Kimio AkagawaJournal of Neurochemistry|April 10, 2016
Unusual social behavior in HPC-1/syntaxin1A knockout mice is caused by disruption of the oxytocinergic neural systemTomonori Fujiwara, Masumi Sanada, Takefumi Kofuji, et al.The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|January 6, 2012
Impairment of catecholamine systems during induction of long-term potentiation at hippocampal CA1 synapses in HPC-1/syntaxin 1A knock-out miceTatsuya Mishima, Tomonori Fujiwara, Takefumi Kofuji, et al.The European Journal of Neuroscience|November 16, 2017
Syntaxin 1B contributes to regulation of the dopaminergic system through GABA transmission in the CNSTomonori Fujiwara, Takefumi Kofuji, Tatsuya Mishima, et al.The European Journal of Neuroscience|June 26, 2010
HPC-1/syntaxin 1A gene knockout mice show abnormal behavior possibly related to a disruption in 5-HTergic systemsTomonori Fujiwara, Masumi Snada, Takefumi Kofuji, et al.Journal of Neurochemistry|March 27, 2014
HPC-1/syntaxin 1A and syntaxin 1B play distinct roles in neuronal survivalTakefumi Kofuji, Tomonori Fujiwara, Masumi Sanada, et al.Frontiers in Neural Circuits|January 6, 2022
Manganese Dynamics in Mouse Brain After Systemic MnCl2 Administration for Activation-Induced Manganese-Enhanced MRIHiroki Tanihira, Tomonori Fujiwara, Satomi Kikuta, et al.Journal of Molecular Histology|August 9, 2011
Unusual retinal layer organization in HPC-1/syntaxin 1A knockout miceYuko Kaneko, Rie Suge, Tomonori Fujiwara, et al.Neuroscience Letters|February 26, 2017
A part of patients with autism spectrum disorder has haploidy of HPC-1/syntaxin1A gene that possibly causes behavioral disturbance as in experimentally gene ablated miceTakefumi Kofuji, Yuko Hayashi, Tomonori Fujiwara, et al.Pageof 3