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Therapeutic Advances in Hematology
|
June 17, 2024
Integrated genetic, epigenetic, and immune landscape of <i>TP53</i> mutant AML and higher risk MDS treated with azacitidine
Amer M Zeidan, Jan Philipp Bewersdorf, Vanessa Hasle, et al.
Haematologica
|
February 13, 2008
NPM1 mutations and cytoplasmic nucleophosmin are mutually exclusive of recurrent genetic abnormalities: a comparative analysis of 2562 patients with acute myeloid leukemia
Brunangelo Falini, Cristina Mecucci, Giuseppe Saglio, et al.
Annals of Hematology
|
March 21, 2020
A 19-year-old patient with atypical chronic myeloid leukemia
Philipp Ernst, Björn Engmann, Jochen J Frietsch, et al.
Haematologica
|
June 29, 2014
Response to azacitidine is independent of p53 expression in higher-risk myelodysplastic syndromes and secondary acute myeloid leukemia
Catharina Müller-Thomas, Martina Rudelius, Ina-Christine Rondak, et al.
Experimental Hematology
|
February 5, 2022
Acute myeloid leukemia exhibiting clonal instability during treatment: Implications for measurable residual disease assessments
Anita T Simonsen, Manja Meggendorfer, Marcus H Hansen, et al.
Seminars in Cancer Biology
|
June 27, 2021
Clinical utility of whole-genome sequencing in precision oncology
Richard Rosenquist, Edwin Cuppen, Reinhard Buettner, et al.
Blood
|
August 25, 2012
SRSF2 mutations in 275 cases with chronic myelomonocytic leukemia (CMML)
Manja Meggendorfer, Andreas Roller, Torsten Haferlach, et al.
Annals of Hematology
|
September 27, 2012
Activating CBL mutations are associated with a distinct MDS/MPN phenotype
Juliana Schwaab, Thomas Ernst, Philipp Erben, et al.
Blood
|
May 16, 2009
Microarray-based classifiers and prognosis models identify subgroups with distinct clinical outcomes and high risk of AML transformation of myelodysplastic syndrome
Ken I Mills, Alexander Kohlmann, P Mickey Williams, et al.
Blood
|
August 24, 2012
A novel hierarchical prognostic model of AML solely based on molecular mutations
Vera Grossmann, Susanne Schnittger, Alexander Kohlmann, et al.
Page
of 60
Search research articles
Search
Showing results (371-380 of 598) with videos related to
Sort By:
Page
of 60
Therapeutic Advances in Hematology
|
June 17, 2024
Integrated genetic, epigenetic, and immune landscape of <i>TP53</i> mutant AML and higher risk MDS treated with azacitidine
Amer M Zeidan, Jan Philipp Bewersdorf, Vanessa Hasle, et al.
Haematologica
|
February 13, 2008
NPM1 mutations and cytoplasmic nucleophosmin are mutually exclusive of recurrent genetic abnormalities: a comparative analysis of 2562 patients with acute myeloid leukemia
Brunangelo Falini, Cristina Mecucci, Giuseppe Saglio, et al.
Annals of Hematology
|
March 21, 2020
A 19-year-old patient with atypical chronic myeloid leukemia
Philipp Ernst, Björn Engmann, Jochen J Frietsch, et al.
Haematologica
|
June 29, 2014
Response to azacitidine is independent of p53 expression in higher-risk myelodysplastic syndromes and secondary acute myeloid leukemia
Catharina Müller-Thomas, Martina Rudelius, Ina-Christine Rondak, et al.
Experimental Hematology
|
February 5, 2022
Acute myeloid leukemia exhibiting clonal instability during treatment: Implications for measurable residual disease assessments
Anita T Simonsen, Manja Meggendorfer, Marcus H Hansen, et al.
Seminars in Cancer Biology
|
June 27, 2021
Clinical utility of whole-genome sequencing in precision oncology
Richard Rosenquist, Edwin Cuppen, Reinhard Buettner, et al.
Blood
|
August 25, 2012
SRSF2 mutations in 275 cases with chronic myelomonocytic leukemia (CMML)
Manja Meggendorfer, Andreas Roller, Torsten Haferlach, et al.
Annals of Hematology
|
September 27, 2012
Activating CBL mutations are associated with a distinct MDS/MPN phenotype
Juliana Schwaab, Thomas Ernst, Philipp Erben, et al.
Blood
|
May 16, 2009
Microarray-based classifiers and prognosis models identify subgroups with distinct clinical outcomes and high risk of AML transformation of myelodysplastic syndrome
Ken I Mills, Alexander Kohlmann, P Mickey Williams, et al.
Blood
|
August 24, 2012
A novel hierarchical prognostic model of AML solely based on molecular mutations
Vera Grossmann, Susanne Schnittger, Alexander Kohlmann, et al.
Page
of 60