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Toru Fukuda

Showing results (61-70 of 68) with videos related to

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The EMBO Journal|June 13, 2014
MiR-133 promotes cardiac reprogramming by directly repressing Snai1 and silencing fibroblast signaturesNaoto Muraoka, Hiroyuki Yamakawa, Kazutaka Miyamoto, et al.
Proceedings of the National Academy of Sciences of the United States of America|May 28, 2009
Distinct function of 2 chromatin remodeling complexes that share a common subunit, Williams syndrome transcription factor (WSTF)Kimihiro Yoshimura, Hirochika Kitagawa, Ryoji Fujiki, et al.
FEBS Letters|May 22, 2014
Netrin-4 derived from murine vascular endothelial cells inhibits osteoclast differentiation in vitro and prevents bone loss in vivoYuichiro Enoki, Tsuyoshi Sato, Shinya Tanaka, et al.
Endocrinology|February 8, 2013
Vitamin D receptor in osteoblasts is a negative regulator of bone mass controlYoko Yamamoto, Tatsuya Yoshizawa, Toru Fukuda, et al.
Biochemical and Biophysical Research Communications|October 28, 2008
A unique mutation of ALK2, G356D, found in a patient with fibrodysplasia ossificans progressiva is a moderately activated BMP type I receptorToru Fukuda, Kazuhiro Kanomata, Junya Nojima, et al.
Proceedings of the National Academy of Sciences of the United States of America|January 23, 2014
Retraction for Yoshimura et al., Distinct function of 2 chromatin remodeling complexes that share a common subunit, Williams syndrome transcription factor (WSTF)Kimihiro Yoshimura, Hirochika Kitagawa, Ryoji Fujiki, et al.
The Journal of Clinical Investigation|June 6, 2007
Strain-dependent embryonic lethality and exaggerated vascular remodeling in heparin cofactor II-deficient miceKen-ichi Aihara, Hiroyuki Azuma, Masashi Akaike, et al.
The Journal of Biological Chemistry|August 8, 2008
Constitutively activated ALK2 and increased SMAD1/5 cooperatively induce bone morphogenetic protein signaling in fibrodysplasia ossificans progressivaToru Fukuda, Masakazu Kohda, Kazuhiro Kanomata, et al.
Pageof 7

Showing results (61-70 of 68) with videos related to

Sort By:
Pageof 7
You have reached the last page of results.This site can display upto 68 results.
The EMBO Journal|June 13, 2014
MiR-133 promotes cardiac reprogramming by directly repressing Snai1 and silencing fibroblast signaturesNaoto Muraoka, Hiroyuki Yamakawa, Kazutaka Miyamoto, et al.
Proceedings of the National Academy of Sciences of the United States of America|May 28, 2009
Distinct function of 2 chromatin remodeling complexes that share a common subunit, Williams syndrome transcription factor (WSTF)Kimihiro Yoshimura, Hirochika Kitagawa, Ryoji Fujiki, et al.
FEBS Letters|May 22, 2014
Netrin-4 derived from murine vascular endothelial cells inhibits osteoclast differentiation in vitro and prevents bone loss in vivoYuichiro Enoki, Tsuyoshi Sato, Shinya Tanaka, et al.
Endocrinology|February 8, 2013
Vitamin D receptor in osteoblasts is a negative regulator of bone mass controlYoko Yamamoto, Tatsuya Yoshizawa, Toru Fukuda, et al.
Biochemical and Biophysical Research Communications|October 28, 2008
A unique mutation of ALK2, G356D, found in a patient with fibrodysplasia ossificans progressiva is a moderately activated BMP type I receptorToru Fukuda, Kazuhiro Kanomata, Junya Nojima, et al.
Proceedings of the National Academy of Sciences of the United States of America|January 23, 2014
Retraction for Yoshimura et al., Distinct function of 2 chromatin remodeling complexes that share a common subunit, Williams syndrome transcription factor (WSTF)Kimihiro Yoshimura, Hirochika Kitagawa, Ryoji Fujiki, et al.
The Journal of Clinical Investigation|June 6, 2007
Strain-dependent embryonic lethality and exaggerated vascular remodeling in heparin cofactor II-deficient miceKen-ichi Aihara, Hiroyuki Azuma, Masashi Akaike, et al.
The Journal of Biological Chemistry|August 8, 2008
Constitutively activated ALK2 and increased SMAD1/5 cooperatively induce bone morphogenetic protein signaling in fibrodysplasia ossificans progressivaToru Fukuda, Masakazu Kohda, Kazuhiro Kanomata, et al.
Pageof 7