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Trudy McGarry

Showing results (11-20 of 26) with videos related to

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Rheumatology (Oxford, England)|November 18, 2022
Rheumatoid arthritis macrophages are primed for inflammation and display bioenergetic and functional alterationsMegan M Hanlon, Trudy McGarry, Viviana Marzaioli, et al.
Journal of Autoimmunity|September 6, 2018
Altered expression of microRNA-23a in psoriatic arthritis modulates synovial fibroblast pro-inflammatory mechanisms via phosphodiesterase 4BSarah M Wade, Michelle Trenkmann, Trudy McGarry, et al.
Arthritis & Rheumatology (Hoboken, N.J.)|May 24, 2018
JAK/STAT Blockade Alters Synovial Bioenergetics, Mitochondrial Function, and Proinflammatory Mediators in Rheumatoid ArthritisTrudy McGarry, Carl Orr, Sarah Wade, et al.
Annals of the Rheumatic Diseases|January 11, 2019
Association of synovial tissue polyfunctional T-cells with DAPSA in psoriatic arthritisSarah M Wade, Mary Canavan, Trudy McGarry, et al.
Annals of the Rheumatic Diseases|August 21, 2015
Acute serum amyloid A is an endogenous TLR2 ligand that mediates inflammatory and angiogenic mechanismsMary Connolly, Peter R Rooney, Trudy McGarry, et al.
Frontiers in Immunology|March 22, 2021
Serum miRNA Signature in Rheumatoid Arthritis and "At-Risk Individuals"Clare C Cunningham, Sarah Wade, Achilleas Floudas, et al.
Scientific Reports|February 23, 2017
Resolution of TLR2-induced inflammation through manipulation of metabolic pathways in Rheumatoid ArthritisTrudy McGarry, Monika Biniecka, Wei Gao, et al.
Clinical & Translational Immunology|January 29, 2021
Rheumatoid arthritis CD14<sup>+</sup> monocytes display metabolic and inflammatory dysfunction, a phenotype that precedes clinical manifestation of diseaseTrudy McGarry, Megan M Hanlon, Viviana Marzaioli, et al.
Ebiomedicine|August 31, 2019
Dysregulated miR-125a promotes angiogenesis through enhanced glycolysisSarah M Wade, Nils Ohnesorge, Hayley McLoughlin, et al.
JCI Insight|December 7, 2018
Enriched Cd141+ DCs in the joint are transcriptionally distinct, activated, and contribute to joint pathogenesisMary Canavan, Alice M Walsh, Vipul Bhargava, et al.
Pageof 3

Showing results (11-20 of 26) with videos related to

Sort By:
Pageof 3
Rheumatology (Oxford, England)|November 18, 2022
Rheumatoid arthritis macrophages are primed for inflammation and display bioenergetic and functional alterationsMegan M Hanlon, Trudy McGarry, Viviana Marzaioli, et al.
Journal of Autoimmunity|September 6, 2018
Altered expression of microRNA-23a in psoriatic arthritis modulates synovial fibroblast pro-inflammatory mechanisms via phosphodiesterase 4BSarah M Wade, Michelle Trenkmann, Trudy McGarry, et al.
Arthritis & Rheumatology (Hoboken, N.J.)|May 24, 2018
JAK/STAT Blockade Alters Synovial Bioenergetics, Mitochondrial Function, and Proinflammatory Mediators in Rheumatoid ArthritisTrudy McGarry, Carl Orr, Sarah Wade, et al.
Annals of the Rheumatic Diseases|January 11, 2019
Association of synovial tissue polyfunctional T-cells with DAPSA in psoriatic arthritisSarah M Wade, Mary Canavan, Trudy McGarry, et al.
Annals of the Rheumatic Diseases|August 21, 2015
Acute serum amyloid A is an endogenous TLR2 ligand that mediates inflammatory and angiogenic mechanismsMary Connolly, Peter R Rooney, Trudy McGarry, et al.
Frontiers in Immunology|March 22, 2021
Serum miRNA Signature in Rheumatoid Arthritis and "At-Risk Individuals"Clare C Cunningham, Sarah Wade, Achilleas Floudas, et al.
Scientific Reports|February 23, 2017
Resolution of TLR2-induced inflammation through manipulation of metabolic pathways in Rheumatoid ArthritisTrudy McGarry, Monika Biniecka, Wei Gao, et al.
Clinical & Translational Immunology|January 29, 2021
Rheumatoid arthritis CD14<sup>+</sup> monocytes display metabolic and inflammatory dysfunction, a phenotype that precedes clinical manifestation of diseaseTrudy McGarry, Megan M Hanlon, Viviana Marzaioli, et al.
Ebiomedicine|August 31, 2019
Dysregulated miR-125a promotes angiogenesis through enhanced glycolysisSarah M Wade, Nils Ohnesorge, Hayley McLoughlin, et al.
JCI Insight|December 7, 2018
Enriched Cd141+ DCs in the joint are transcriptionally distinct, activated, and contribute to joint pathogenesisMary Canavan, Alice M Walsh, Vipul Bhargava, et al.
Pageof 3