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Life Sciences|July 22, 2006
Molecular and immunohistochemical studies in expression of voltage-dependent Ca2+ channels in dorsal root ganglia from streptozotocin-induced diabetic miceMariko Umeda, Tsuyako Ohkubo, Jun Ono, et al.Biochemical and Biophysical Research Communications|February 1, 2015
Integrin α6β4 and TRPV1 channel coordinately regulate directional keratinocyte migrationAyako Miyazaki, Tsuyako Ohkubo, Mitsutoki Hatta, et al.Archives of Oral Biology|August 25, 2010
Specific expression of salivary maxi-K channel variant is augmented in diabetic miceKazuhiko Okamura, Kenichi Kato, Ryuji Uchida, et al.Biochemical and Biophysical Research Communications|February 11, 2014
Endogenous and exogenous hydrogen sulfide facilitates T-type calcium channel currents in Cav3.2-expressing HEK293 cellsFumiko Sekiguchi, Yosuke Miyamoto, Daiki Kanaoka, et al.Pain|March 30, 2016
Therapeutic potential of RQ-00311651, a novel T-type Ca2+ channel blocker, in distinct rodent models for neuropathic and visceral painFumiko Sekiguchi, Yuma Kawara, Maho Tsubota, et al.Redox Biology|December 23, 2022
A hydrolysate of poly-trans-[(2-carboxyethyl)germasesquioxane] (Ge-132) suppresses Cav3.2-dependent pain by sequestering exogenous and endogenous sulfideFumiko Sekiguchi, Nene Koike, Yasuhiro Shimada, et al.European Journal of Medicinal Chemistry|September 8, 2022
Discovery of pimozide derivatives as novel T-type calcium channel inhibitors with little binding affinity to dopamine D2 receptors for treatment of somatic and visceral painYoshihito Kasanami, Chihiro Ishikawa, Takahiro Kino, et al.Journal of Pharmacological Sciences|September 8, 2019
Genetic deletion of Cav3.2 T-type calcium channels abolishes H2S-dependent somatic and visceral pain signaling in C57BL/6 miceKazuki Matsui, Maho Tsubota, Saaya Fukushi, et al.Neuropharmacology|June 19, 2018
Blockade of T-type calcium channels by 6-prenylnaringenin, a hop component, alleviates neuropathic and visceral pain in miceFumiko Sekiguchi, Tomoyo Fujita, Takahiro Deguchi, et al.Pageof 2