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U A Boelsterli

Showing results (1-10 of 43) with videos related to

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Praxis|June 1, 2002
[Unexpected liver toxicity of therapeutic drugs: dangerous molecule or hypersensitive patient?]U A Boelsterli
International Journal of Clinical Practice. Supplement|August 9, 2002
Nimesulide and hepatic adverse effects: roles of reactive metabolites and host factorsU A Boelsterli
Drug Metabolism Reviews|January 1, 1993
Specific targets of covalent drug-protein interactions in hepatocytes and their toxicological significance in drug-induced liver injuryU A Boelsterli
Gastroenterology|December 3, 1998
Diclofenac acyl glucuronide, a major biliary metabolite, is directly involved in small intestinal injury in ratsS Seitz, U A Boelsterli
Archives of Toxicology|January 1, 1991
Biomechanisms of cocaine-induced hepatocyte injury mediated by the formation of reactive metabolitesU A Boelsterli, C Göldlin
Xenobiotica; the Fate of Foreign Compounds in Biological Systems|March 1, 1994
Dissociation of covalent protein adduct formation from oxidative injury in cultured hepatocytes exposed to cocaineC Göldlin, U A Boelsterli
Toxicology and Applied Pharmacology|July 1, 1990
Modulation of cocaine metabolism in primary rat hepatocyte cultures: effects on irreversible binding and protein biosynthesisP Bouis, U A Boelsterli
Archives of Toxicology|April 1, 1985
Early biochemical and morphological changes of the rat adrenal medulla induced by xylitolU A Boelsterli, G Zbinden
The American Journal of Physiology|August 8, 1998
The labile iron pool in hepatocytes: prooxidant-induced increase in free iron precedes oxidative cell injuryA Stäubli, U A Boelsterli
Toxicology and Applied Pharmacology|May 1, 1993
Diclofenac covalent protein binding is dependent on acyl glucuronide formation and is inversely related to P450-mediated acute cell injury in cultured rat hepatocytesA Kretz-Rommel, U A Boelsterli
Pageof 5

Showing results (1-10 of 43) with videos related to

Sort By:
Pageof 5
Praxis|June 1, 2002
[Unexpected liver toxicity of therapeutic drugs: dangerous molecule or hypersensitive patient?]U A Boelsterli
International Journal of Clinical Practice. Supplement|August 9, 2002
Nimesulide and hepatic adverse effects: roles of reactive metabolites and host factorsU A Boelsterli
Drug Metabolism Reviews|January 1, 1993
Specific targets of covalent drug-protein interactions in hepatocytes and their toxicological significance in drug-induced liver injuryU A Boelsterli
Gastroenterology|December 3, 1998
Diclofenac acyl glucuronide, a major biliary metabolite, is directly involved in small intestinal injury in ratsS Seitz, U A Boelsterli
Archives of Toxicology|January 1, 1991
Biomechanisms of cocaine-induced hepatocyte injury mediated by the formation of reactive metabolitesU A Boelsterli, C Göldlin
Xenobiotica; the Fate of Foreign Compounds in Biological Systems|March 1, 1994
Dissociation of covalent protein adduct formation from oxidative injury in cultured hepatocytes exposed to cocaineC Göldlin, U A Boelsterli
Toxicology and Applied Pharmacology|July 1, 1990
Modulation of cocaine metabolism in primary rat hepatocyte cultures: effects on irreversible binding and protein biosynthesisP Bouis, U A Boelsterli
Archives of Toxicology|April 1, 1985
Early biochemical and morphological changes of the rat adrenal medulla induced by xylitolU A Boelsterli, G Zbinden
The American Journal of Physiology|August 8, 1998
The labile iron pool in hepatocytes: prooxidant-induced increase in free iron precedes oxidative cell injuryA Stäubli, U A Boelsterli
Toxicology and Applied Pharmacology|May 1, 1993
Diclofenac covalent protein binding is dependent on acyl glucuronide formation and is inversely related to P450-mediated acute cell injury in cultured rat hepatocytesA Kretz-Rommel, U A Boelsterli
Pageof 5