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Naunyn-Schmiedeberg'S Archives of Pharmacology|December 5, 1998
Cardioprotective actions of KC 12291. I. Inhibition of voltage-gated Na+ channels in ischemia delays myocardial Na+ overloadU K Decking, M Hartmann, H Rose, et al.Cardiovascular Research|July 27, 2001
The nitric oxide-induced reduction in cardiac energy supply is not due to inhibition of creatine kinaseU K Decking, J P Williams, R Dahmann, et al.Proceedings of the National Academy of Sciences of the United States of America|September 1, 1999
Disruption of myoglobin in mice induces multiple compensatory mechanismsA Gödecke, U Flögel, K Zanger, et al.Circulation Research|February 19, 1998
Coronary hemodynamics in endothelial NO synthase knockout miceA Gödecke, U K Decking, Z Ding, et al.Pflugers Archiv : European Journal of Physiology|April 11, 2001
Spatial heterogeneity of energy turnover in the heartU K Decking, S Skwirba, M F Zimmermann, et al.Cardiovascular Research|February 17, 1998
Nitric oxide induced contractile dysfunction is related to a reduction in myocardial energy generationM Kelm, S Schäfer, R Dahmann, et al.The Journal of Physiology|May 23, 2001
Inotropic response to beta-adrenergic receptor stimulation and anti-adrenergic effect of ACh in endothelial NO synthase-deficient mouse heartsA Gödecke, T Heinicke, A Kamkin, et al.Pageof 3