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U Mende

Showing results (61-70 of 79) with videos related to

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Molecular and Cellular Biochemistry|April 12, 1996
Protein phosphorylation in isolated trabeculae from nonfailing and failing human heartsS Bartel, B Stein, T Eschenhagen, et al.
European Heart Journal|December 1, 1991
Beta-adrenoceptor stimulation-induced increase in cardiac Gi-protein expression and in carbachol sensitivityT Eschenhagen, U Mende, W Schmitz, et al.
Proceedings of the National Academy of Sciences of the United States of America|March 4, 1997
G alpha(o) is necessary for muscarinic regulation of Ca2+ channels in mouse heartD Valenzuela, X Han, U Mende, et al.
Molecular Pharmacology|November 1, 1992
Long term beta-adrenoceptor-mediated up-regulation of Gi alpha and G(o) alpha mRNA levels and pertussis toxin-sensitive guanine nucleotide-binding proteins in rat heartT Eschenhagen, U Mende, M Diederich, et al.
Circulation Research|April 1, 1992
Increased messenger RNA level of the inhibitory G protein alpha subunit Gi alpha-2 in human end-stage heart failureT Eschenhagen, U Mende, M Nose, et al.
Journal of Molecular and Cellular Cardiology|February 1, 1994
Radioimmunochemical quantification of Gi alpha in right and left ventricles from patients with ischaemic and dilated cardiomyopathy and predominant left ventricular failureM Böhm, T Eschenhagen, P Gierschik, et al.
Deutsche Medizinische Wochenschrift (1946)|July 10, 1987
[Treatment of tumor hypercalcemia with clodronate. Effect on parathormone and calcitriol]S H Scharla, H W Minne, P Sattar, et al.
Der Radiologe|May 2, 2000
[Computer-assisted oral, maxillary and facial surgery]S Hassfeld, J Brief, R Krempien, et al.
Basic Research in Cardiology|January 1, 1992
Regulation and possible functional implications of G-protein mRNA expression in nonfailing and failing ventricular myocardiumT Eschenhagen, U Mende, M Nose, et al.
American Journal of Physiology. Heart and Circulatory Physiology|July 16, 2017
G<sub>q</sub>-activated fibroblasts induce cardiomyocyte action potential prolongation and automaticity in a three-dimensional microtissue environmentC M Kofron, T Y Kim, M E King, et al.
Pageof 8

Showing results (61-70 of 79) with videos related to

Sort By:
Pageof 8
Molecular and Cellular Biochemistry|April 12, 1996
Protein phosphorylation in isolated trabeculae from nonfailing and failing human heartsS Bartel, B Stein, T Eschenhagen, et al.
European Heart Journal|December 1, 1991
Beta-adrenoceptor stimulation-induced increase in cardiac Gi-protein expression and in carbachol sensitivityT Eschenhagen, U Mende, W Schmitz, et al.
Proceedings of the National Academy of Sciences of the United States of America|March 4, 1997
G alpha(o) is necessary for muscarinic regulation of Ca2+ channels in mouse heartD Valenzuela, X Han, U Mende, et al.
Molecular Pharmacology|November 1, 1992
Long term beta-adrenoceptor-mediated up-regulation of Gi alpha and G(o) alpha mRNA levels and pertussis toxin-sensitive guanine nucleotide-binding proteins in rat heartT Eschenhagen, U Mende, M Diederich, et al.
Circulation Research|April 1, 1992
Increased messenger RNA level of the inhibitory G protein alpha subunit Gi alpha-2 in human end-stage heart failureT Eschenhagen, U Mende, M Nose, et al.
Journal of Molecular and Cellular Cardiology|February 1, 1994
Radioimmunochemical quantification of Gi alpha in right and left ventricles from patients with ischaemic and dilated cardiomyopathy and predominant left ventricular failureM Böhm, T Eschenhagen, P Gierschik, et al.
Deutsche Medizinische Wochenschrift (1946)|July 10, 1987
[Treatment of tumor hypercalcemia with clodronate. Effect on parathormone and calcitriol]S H Scharla, H W Minne, P Sattar, et al.
Der Radiologe|May 2, 2000
[Computer-assisted oral, maxillary and facial surgery]S Hassfeld, J Brief, R Krempien, et al.
Basic Research in Cardiology|January 1, 1992
Regulation and possible functional implications of G-protein mRNA expression in nonfailing and failing ventricular myocardiumT Eschenhagen, U Mende, M Nose, et al.
American Journal of Physiology. Heart and Circulatory Physiology|July 16, 2017
G<sub>q</sub>-activated fibroblasts induce cardiomyocyte action potential prolongation and automaticity in a three-dimensional microtissue environmentC M Kofron, T Y Kim, M E King, et al.
Pageof 8