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Experimental & Molecular Medicine|June 29, 2022
Structural, molecular, and functional insights into Schlafen proteinsUkhyun Jo, Yves PommierCancers|September 28, 2021
Precision Oncology with Drugs Targeting the Replication Stress, ATR, and Schlafen 11Ukhyun Jo, Yasuhisa Murai, Naoko Takebe, et al.DNA Repair|July 17, 2020
Debulking of topoisomerase DNA-protein crosslinks (TOP-DPC) by the proteasome, non-proteasomal and non-proteolytic pathwaysYilun Sun, Liton Kumar Saha, Sourav Saha, et al.Iscience|October 25, 2021
Schlafen 11 expression in human acute leukemia cells with gain-of-function mutations in the interferon-JAK signaling pathwayYasuhisa Murai, Ukhyun Jo, Junko Murai, et al.Nucleic Acids Research|September 22, 2021
Replication-dependent cytotoxicity and Spartan-mediated repair of trapped PARP1-DNA complexesLiton Kumar Saha, Yasuhisa Murai, Sourav Saha, et al.Nature Communications|April 6, 2023
Replication-associated formation and repair of human topoisomerase IIIα cleavage complexesLiton Kumar Saha, Sourav Saha, Xi Yang, et al.Cell Reports|March 27, 2020
Chromatin Remodeling and Immediate Early Gene Activation by SLFN11 in Response to Replication StressJunko Murai, Hongliang Zhang, Lorinc Pongor, et al.Proceedings of the National Academy of Sciences of the United States of America|February 4, 2021
SLFN11 promotes CDT1 degradation by CUL4 in response to replicative DNA damage, while its absence leads to synthetic lethality with ATR/CHK1 inhibitorsUkhyun Jo, Yasuhisa Murai, Sirisha Chakka, et al.Molecular Cancer Therapeutics|May 22, 2023
Schlafen 11 (SLFN11) Kills Cancer Cells Undergoing Unscheduled Re-replicationJunko Murai, Michele Ceribelli, Haiqing Fu, et al.Molecules and Cells|July 22, 2015
Exploiting the Fanconi Anemia Pathway for Targeted Anti-Cancer TherapyUkhyun Jo, Hyungjin KimPageof 63