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Thrombosis and Haemostasis|July 30, 2011
Deletion of human GP1BB and SEPT5 is associated with Bernard-Soulier syndrome, platelet secretion defect, polymicrogyria, and developmental delayIngrid Bartsch, Kirstin Sandrock, Francois Lanza, et al.
Early Human Development|April 19, 2020
General Movement Assessment from videos of computed 3D infant body models is equally effective compared to conventional RGB video ratingA Sebastian Schroeder, Nikolas Hesse, Raphael Weinberger, et al.
Acta Paediatrica (Oslo, Norway : 1992)|December 22, 2025
Infants' General Movements Were Not Affected by Exposure to Maternal Severe Acute Respiratory Syndrome Coronavirus 2 InfectionsKathrin Neumayr, Katharina Lippert, A Sebastian Schroeder, et al.
Paediatric Respiratory Reviews|October 12, 2002
The development of childhood asthma: lessons from the German Multicentre Allergy Study (MAS)Susanne Lau, Renate Nickel, Bodo Niggemann, et al.
European Journal of Paediatric Neurology : EJPN : Official Journal of the European Paediatric Neurology Society|September 10, 2013
Epilepsy in Aicardi-Goutières syndromeGeorgia Ramantani, Louis G Maillard, Thomas Bast, et al.
Journal of the Neurological Sciences|January 26, 2023
Neurological soft signs are associated with reduced medial-lateral postural control in adolescent athletesElena M Bonke, Amanda Clauwaert, Stefan M Hillmann, et al.
Cerebral Cortex (New York, N.Y. : 1991)|November 25, 2022
Neurological soft signs in adolescents are associated with brain structureElena M Bonke, Michaela V Bonfert, Stefan M Hillmann, et al.
Nature Genetics|July 18, 2006
Mutations in genes encoding ribonuclease H2 subunits cause Aicardi-Goutières syndrome and mimic congenital viral brain infectionYanick J Crow, Andrea Leitch, Bruce E Hayward, et al.
American Journal of Human Genetics|September 12, 2007
Clinical and molecular phenotype of Aicardi-Goutieres syndromeGillian Rice, Teresa Patrick, Rekha Parmar, et al.
American Journal of Medical Genetics. Part A|January 22, 2015
Characterization of human disease phenotypes associated with mutations in TREX1, RNASEH2A, RNASEH2B, RNASEH2C, SAMHD1, ADAR, and IFIH1Yanick J Crow, Diana S Chase, Johanna Lowenstein Schmidt, et al.
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