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Blood|September 15, 2007
Simultaneous loss of beta- and gamma-catenin does not perturb hematopoiesis or lymphopoiesisUte Koch, Anne Wilson, Monica Cobas, et al.Cancer Cell|October 20, 2012
Loss of cutaneous TSLP-dependent immune responses skews the balance of inflammation from tumor protective to tumor promotingMatteo Di Piazza, Craig S Nowell, Ute Koch, et al.European Journal of Immunology|July 25, 2013
DL4-mediated Notch signaling is required for the development of fetal αβ and γδ T cellsIsabel Ferrero, Ute Koch, Stephanie Claudinot, et al.Blood|January 29, 2021
Notch signaling promotes disease initiation and progression in murine chronic lymphocytic leukemiaDelphine Tardivon, Mateusz Antoszewski, Nadine Zangger, et al.Nature Communications|September 7, 2017
Signalling strength determines proapoptotic functions of STINGMuhammet F Gulen, Ute Koch, Simone M Haag, et al.Immunity|November 25, 2010
Hes1 is a critical but context-dependent mediator of canonical Notch signaling in lymphocyte development and transformationAgnieszka A Wendorff, Ute Koch, F Thomas Wunderlich, et al.Plos Pathogens|March 8, 2012
Redundant Notch1 and Notch2 signaling is necessary for IFNγ secretion by T helper 1 cells during infection with Leishmania majorFloriane Auderset, Steffen Schuster, Manuel Coutaz, et al.Innate Immunity|May 27, 2010
Factors determining the spontaneous activation of splenic dendritic cells in cultureDavid Vremec, Meredith O'Keeffe, Anne Wilson, et al.FEBS Letters|April 1, 2006
Multiple functions of Notch signaling in self-renewing organs and cancerAnne Wilson, Freddy RadtkeJournal of Immunology (Baltimore, Md. : 1950)|June 22, 2016
Notch Signaling Regulates the Homeostasis of Tissue-Restricted Innate-like T CellsVijaykumar Chennupati, Ute Koch, Manuel Coutaz, et al.Pageof 16