Showing results (71-80 of 82) with videos related to
Sort By:
Pageof 9
Frontiers in Pharmacology|October 7, 2017
The Effects of SEA0400 on Ca2+ Transient Amplitude and Proarrhythmia Depend on the Na+/Ca2+ Exchanger Expression Level in Murine ModelsNils Bögeholz, Jan S Schulte, Sven Kaese, et al.Frontiers in Pharmacology|February 18, 2021
Initial Characterization of Stressed Transgenic Mice With Cardiomyocyte-Specific Overexpression of Protein Phosphatase 2CPaula Bollmann, Franziska Werner, Marko Jaron, et al.Frontiers in Pharmacology|May 28, 2021
Histamine can be Formed and Degraded in the Human and Mouse HeartJoachim Neumann, Juliane M Grobe, Jacqueline Weisgut, et al.The Journal of Biological Chemistry|August 13, 2022
Impaired myocellular Ca2+ cycling in protein phosphatase PP2A-B56α KO mice is normalized by β-adrenergic stimulationDennis Glaser, Alexander Heinick, Julius R Herting, et al.Journal of Muscle Research and Cell Motility|October 1, 2011
Protein phosphatase 2A affects myofilament contractility in non-failing but not in failing human myocardiumPaul J M Wijnker, Peter Boknik, Ulrich Gergs, et al.FASEB Journal : Official Publication of the Federation of American Societies for Experimental Biology|May 30, 2015
Annexin A4 is a novel direct regulator of adenylyl cyclase type 5Alexander Heinick, Xenia Husser, Kirsten Himmler, et al.Frontiers in Pharmacology|October 18, 2019
Evidence for Arrhythmogenic Effects of A2A-Adenosine ReceptorsPeter Boknik, Katharina Drzewiecki, John Eskandar, et al.The Journal of Pharmacology and Experimental Therapeutics|March 26, 2024
Initial Characterization of a Transgenic Mouse with Overexpression of the Human H1-Histamine Receptor on the HeartLina Maria Rayo Abella, Hannes Jacob, Max Keller, et al.Heart Rhythm|August 24, 2010
Knock-in gain-of-function sodium channel mutation prolongs atrial action potentials and alters atrial vulnerabilityAndreas Blana, Sven Kaese, Lisa Fortmüller, et al.Circulation. Heart Failure|November 27, 2014
First report on an inotropic peptide activating tetrodotoxin-sensitive, "neuronal" sodium currents in the heartPaulus Kirchhof, Tzachy Tal, Larissa Fabritz, et al.Pageof 9