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American Journal of Physiology. Heart and Circulatory Physiology|July 19, 2000
Basic FGF reduces stunning via a NOS2-dependent pathway in coronary-perfused mouse heartsT G Hampton, I Amende, J Fong, et al.Circulation Research|September 29, 2000
iNOS gene expression modulates microvascular responsiveness in endotoxin-challenged miceW A Boyle, L S Parvathaneni, V Bourlier, et al.Shock (Augusta, Ga.)|November 9, 2001
The roles of iNOS in liver ischemia-reperfusion injuryV G Lee, M L Johnson, J Baust, et al.American Journal of Transplantation : Official Journal of the American Society of Transplantation and the American Society of Transplant Surgeons|July 20, 2013
Receptor for advanced glycation end products (RAGE) on iNKT cells mediates lung ischemia-reperfusion injuryA K Sharma, D J LaPar, M L Stone, et al.The Journal of Surgical Research|August 18, 2000
Deletion of endothelial nitric oxide synthase exacerbates myocardial stunning in an isolated mouse heart modelR L Hannan, M C John, P C Kouretas, et al.British Journal of Pharmacology|March 14, 2007
Interactions between NO, CO and an endothelium-derived hyperpolarizing factor (EDHF) in maintaining patency of the ductus arteriosus in the mouseB Baragatti, F Brizzi, S Barogi, et al.Journal of Leukocyte Biology|April 1, 1996
Transcriptional basis for hyporesponsiveness of the human inducible nitric oxide synthase gene to lipopolysaccharide/interferon-gammaX Zhang, V E Laubach, E W Alley, et al.Surgery|August 24, 1999
Inducible nitric oxide synthase (iNOS) gene deficiency increases the mortality of sepsis in miceJ P Cobb, R S Hotchkiss, P E Swanson, et al.American Journal of Physiology. Lung Cellular and Molecular Physiology|August 25, 2000
Attenuation of lung reperfusion injury after transplantation using an inhibitor of nuclear factor-kappaBS D Ross, I L Kron, J J Gangemi, et al.The Annals of Thoracic Surgery|June 1, 2001
Retinoic acid enhances lung growth after pneumonectomyA K Kaza, I L Kron, J A Kern, et al.Pageof 3