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International Immunology|November 5, 1999
Molecular mechanisms of T lymphocyte activation: convergence of T cell antigen receptor and IL-1 receptor-induced signaling at the level of IL-2 gene transcriptionG Varga, U Dreikhausen, M Kracht, et al.
FEBS Letters|July 12, 1993
Coexpression of type I and type II IL-1 receptors in the murine T helper 2 cell line D10NR Brigelius-Flohé, J E McCarthy, K Resch, et al.
Prostaglandins|September 1, 1986
Control of prostanoid synthesis: role of reincorporation of released precursor fatty acidsM Goppelt-Struebe, C F Koerner, G Hausmann, et al.
Biochemical Pharmacology|September 15, 1990
Glucocorticoid-induced reduction of prostanoid synthesis in TPA-differentiated U937 cells is mainly due to a reduced cyclooxygenase activityL Koehler, R Hass, D L DeWitt, et al.
Biochimica Et Biophysica Acta|May 29, 1987
Separation of plasma membrane domains of calf thymocytes by affinity chromatography on ouabain-SepharoseM Szamel, M Goppelt-Strübe, W Bessler, et al.
Journal of Immunology (Baltimore, Md. : 1950)|April 15, 1987
Activation of glomerular mesangial cells by the terminal membrane attack complex of complementD H Lovett, G M Haensch, M Goppelt, et al.
European Journal of Immunology|July 1, 1994
Interleukin-1-induced activation of a protein kinase co-precipitating with the type I interleukin-1 receptor in T cellsM Martin, G F Böl, A Eriksson, et al.
The Journal of Biological Chemistry|November 10, 2000
The NF-kappa b repressing factor is involved in basal repression and interleukin (IL)-1-induced activation of IL-8 transcription by binding to a conserved NF-kappa b-flanking sequence elementM Nourbakhsh, S Kalble, A Dorrie, et al.
FEBS Letters|February 14, 1998
Protein binding regions of the mRNAs for the 55 kDa tumor necrosis factor receptor and the glucose transporter 1: sequence homology and competition for cellular proteinsS Kafert, R Winzen, A Loos, et al.
The Journal of Biological Chemistry|March 21, 1997
The interleukin-1 receptor accessory protein (IL-1RAcP) is essential for IL-1-induced activation of interleukin-1 receptor-associated kinase (IRAK) and stress-activated protein kinases (SAP kinases)H Wesche, C Korherr, M Kracht, et al.
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