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Proceedings of the National Academy of Sciences of the United States of America|March 22, 2006
Properties of human connexin 31, which is implicated in hereditary dermatological disease and deafnessCharles K Abrams, Mona M Freidin, Vytas K Verselis, et al.Proceedings of the National Academy of Sciences of the United States of America|October 18, 2013
Fast structural responses of gap junction membrane domains to AB5 toxinsIrina V Majoul, Liang Gao, Eric Betzig, et al.Journal of Neuroinflammation|August 6, 2022
Microglia-specific deletion of histone deacetylase 3 promotes inflammation resolution, white matter integrity, and functional recovery in a mouse model of traumatic brain injuryYongfang Zhao, Hongfeng Mu, Yichen Huang, et al.Neuropharmacology|April 16, 2013
The ATP required for potentiation of skeletal muscle contraction is released via pannexin hemichannelsManuel A Riquelme, Luis A Cea, José L Vega, et al.The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|March 27, 2003
Ischemic insults derepress the gene silencer REST in neurons destined to dieAgata Calderone, Teresa Jover, Kyung-min Noh, et al.Molecular Biosystems|January 6, 2012
Modulation of gap junction channels and hemichannels by growth factorsKurt A Schalper, Manuel A Riquelme, María C Brañes, et al.Proceedings of the National Academy of Sciences of the United States of America|January 5, 2002
Metabolic inhibition induces opening of unapposed connexin 43 gap junction hemichannels and reduces gap junctional communication in cortical astrocytes in cultureJorge E Contreras, Helmut A Sánchez, Eliseo A Eugenin, et al.Proceedings of the National Academy of Sciences of the United States of America|March 19, 2008
Ischemic preconditioning blocks BAD translocation, Bcl-xL cleavage, and large channel activity in mitochondria of postischemic hippocampal neuronsTakahiro Miyawaki, Toshihiro Mashiko, Dimitry Ofengeim, et al.The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|August 19, 2022
STAT1 Contributes to Microglial/Macrophage Inflammation and Neurological Dysfunction in a Mouse Model of Traumatic Brain InjuryYongfang Zhao, Cheng Ma, Caixia Chen, et al.Antioxidants & Redox Signaling|September 26, 2008
Modulation of brain hemichannels and gap junction channels by pro-inflammatory agents and their possible role in neurodegenerationJuan A Orellana, Pablo J Sáez, Kenji F Shoji, et al.Pageof 9