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Oncogene|July 13, 2010
Heat-shock transcription factor HSF1 has a critical role in human epidermal growth factor receptor-2-induced cellular transformation and tumorigenesisL Meng, V L Gabai, M Y ShermanTissue & Cell|January 1, 1992
Association of blebbing with assembly of cytoskeletal proteins in ATP-depleted EL-4 ascites tumour cellsV L Gabai, A E Kabakov, A F MosinFEBS Letters|January 3, 1994
Mitochondrial ATP hydrolysis and ATP depletion in thymocytes and Ehrlich ascites carcinoma cellsB V Chernyak, V N Dedov, V L GabaiOncogene|January 12, 2010
HSP72 depletion suppresses gammaH2AX activation by genotoxic stresses via p53/p21 signalingV L Gabai, M Y Sherman, J A YaglomBiochemistry. Biokhimiia|May 9, 2002
Necrosis is an active and controlled form of programmed cell deathS Ya Proskuryakov, V L Gabai, A G KonoplyannikovFEBS Letters|November 24, 1999
ATPase activity of the heat shock protein hsp72 is dispensable for its effects on dephosphorylation of stress kinase JNK and on heat-induced apoptosisV Volloch, V L Gabai, S Rits, et al.Biomedical Science|April 1, 1990
Oxidative stress, disturbance of energy balance, and death of ascites tumour cells under menadione (vitamin K3) actionV L Gabai, A S Seilanov, Makarova YuM, et al.FEBS Letters|August 26, 1996
Distinct effects of heat shock and ATP depletion on distribution and isoform patterns of human Hsp27 in endothelial cellsS A Loktionova, O P Ilyinskaya, V L Gabai, et al.Biochemistry and Molecular Biology International|January 1, 1995
Spontaneous overexpression of heat-shock proteins in Ehrlich ascites carcinoma cells during in vivo growthV L Gabai, V A Mosina, K R Budagova, et al.Cell Stress & Chaperones|January 9, 2001
HSP72 can protect cells from heat-induced apoptosis by accelerating the inactivation of stress kinase JNKV Volloch, V L Gabai, S Rits, et al.Pageof 4