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Blood|May 15, 1993
Mechanism of differential inhibition of factor-dependent cell proliferation by transforming growth factor-beta 1: selective uncoupling of FMS from MYCA R Chen, L R RohrschneiderJournal of Virology|August 1, 1984
Common features of the yes and src gene products defined by peptide-specific antibodiesL E Gentry, L R RohrschneiderMolecular and Cellular Biology|March 1, 1983
Increased phosphorylation of vinculin on tyrosine does not occur during the release of stress fibers before mitosis in normal cellsM J Rosok, L R RohrschneiderBlood|March 9, 1999
A novel spliced form of SH2-containing inositol phosphatase is expressed during myeloid developmentD M Lucas, L R RohrschneiderCell|November 30, 1990
Expression of the M-CSF receptor is controlled posttranscriptionally by the dominant actions of GM-CSF or multi-CSFB C Gliniak, L R RohrschneiderOncogene Research|January 1, 1989
The kinase activity of the v-fms encoded protein has a low pH optimumS D Lyman, L R RohrschneiderCell|December 23, 1988
Activation of the feline c-fms proto-oncogene: multiple alterations are required to generate a fully transformed phenotypeJ Woolford, A McAuliffe, L R RohrschneiderOncogene|October 1, 1988
Colony stimulating factor-1 induced growth stimulation of v-fms transformed fibroblastsS D Lyman, L Park, L R RohrschneiderCell|June 1, 1982
Nuclear location of the putative transforming protein of avian myelocytomatosis virusH D Abrams, L R Rohrschneider, R N EisenmanBlood|February 15, 1994
Induced expression of c-fms in normal hematopoietic cells shows evidence for both conservation and lineage restriction of signal transduction in response to macrophage colony-stimulating factorG A McArthur, L R Rohrschneider, G R JohnsonPageof 6